ReviewInternational journal of biological sciences2026
New Insights into the Role of NF-κB in Hepatitis B Virus Infection.
Review in International journal of biological sciences, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
1 citing paper in PubMed.
- MicroRNA Control of Hepatocyte-Stromal Crosstalk in the Early Premalignant Microenvironment of HBV-Associated HCC.International journal of molecular sciences · 2026Review
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
8 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
The nuclear factor kappaB (NF-κB) is a critical regulator in immune development, responses, and tumorigenesis. A range of signals can stimulate its activation by interacting with the inhibitor of κB (IκB) and IκB kinase (IKK) through the canonical pathway or non-canonical pathway. Activation of NF-κB results in this transcriptional factor binding with specific κB sites of different target genes. The NF-κB signaling pathway is particularly associated with liver diseases including hepatitis B virus (HBV) infection. HBV proteins have been shown to interact with different members of the NF-κB system, either enhancing or suppressing its activity. Here, we summarize current understanding on the interaction between HBV proteins and the NF-κB pathway, along with its association with liver diseases and potential therapeutic targets.
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Registered trials
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