Evidence map›Paper›PMID 41800241›Full record

ArticleInternational journal of biological sciences2026

Exploring Host-driven Immunopathological Factors Developing Severe Tuberculosis: Insights from Comparative Mouse Models.

Hongmin Kim, Kee Woong Kwon, Hagyu Kim, WeonSeok Jung, Kyungmin Kim, Jung Joo Hong, Sung Jae Shin

Abstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in International journal of biological sciences, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Hongmin KimDepartment of Microbiology, Institute for Immunology and Immunological Disease, Yonsei University College of Medicine, Seoul, South Korea.
Kee Woong KwonDepartment of Microbiology, Institute for Immunology and Immunological Disease, Yonsei University College of Medicine, Seoul, South Korea.
Hagyu KimDepartment of Microbiology, Graduate School of Medical Science, Brain Korea 21 Project, Yonsei University College of Medicine, Seoul, South Korea.
WeonSeok JungDepartment of Microbiology, Graduate School of Medical Science, Brain Korea 21 Project, Yonsei University College of Medicine, Seoul, South Korea.
Kyungmin KimDepartment of Microbiology, Graduate School of Medicine, Yonsei University College of Medicine, Seoul, South Korea.
Jung Joo HongNational Primate Research Center, Korea Research Institute of Bioscience and Biotechnology (KRIBB), Cheongju, Chungcheongbuk, South Korea.
Sung Jae ShinDepartment of Microbiology, Institute for Immunology and Immunological Disease, Yonsei University College of Medicine, Seoul, South Korea.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Tuberculosis (TB) pathogenesis arises from complex interactions between host immune responses and the genetic diversity of

Indexed as

Mycobacterium tuberculosisTuberculosisAnimalsDisease Models, AnimalFemaleGranulocyte Colony-Stimulating FactorLungMiceMice, Inbred C3HMice, Inbred C57BLNeutrophilsGranulocyte Colony-Stimulating Factorhost susceptibilityinbred mouse modelsmycobacterium tuberculosisneutrophil-to-T cell ratiotype I interferon

Identifiers

PMID41800241
PMCPMC12965220

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.