Evidence map›Paper›PMID 41800133›Full record

ReviewPeerJ2026

Clinical features and advances in the genetics of periodic paralysis.

Man Luo, Beibei Liu, Junjie Xu, Danyang Meng

Abstract readReview
PubMed Full text at PMC Publisher
In one paragraph

Review in PeerJ, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Permanent weakness and myopathy in hypokalemic periodic paralysis.Acta myologica : myopathies and cardiomyopathies : official journal of the Mediterranean Society of Myology · 2026
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Man LuoDepartment of Neurology, Affiliated Hospital of Jiaxing University, Jiaxing, Zhejiang, China.
Beibei LiuDepartment of Central Laboratory, Affiliated Hospital of Jiaxing University, Jiaxing, Zhejiang, China.
Junjie XuDepartment of Neurology, Affiliated Hospital of Jiaxing University, Jiaxing, Zhejiang, China.
Danyang MengDepartment of Neurology, Affiliated Hospital of Jiaxing University, Jiaxing, Zhejiang, China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Periodic paralysis (PP) is a group of ion channel diseases with incomplete autosomal dominant inheritance, except in sporadic patients. Ion channel gene mutations cause transient abnormalities in skeletal muscle excitability and muscle weakness. Different mutation sites cause different pathogenesis, which is very important for the classification, clinical manifestations, treatment and prognosis of periodic paralysis. Currently, the recognized mutated genes are

Indexed as

Paralyses, Familial PeriodicCalcium ChannelsCalcium Channels, L-TypeHumansMutationNAV1.4 Voltage-Gated Sodium ChannelPotassium Channels, Inwardly RectifyingCACNA1S protein, humanCalcium ChannelsCalcium Channels, L-TypeNAV1.4 Voltage-Gated Sodium ChannelPotassium Channels, Inwardly RectifyingSCN4A protein, humanClinical featureIon channelopathyMutant geneParoxysmal muscle weaknessPeriodic paralysis

Identifiers

PMID41800133
PMCPMC12965173

What OpenQuestion holds

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LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.