Evidence map›Paper›PMID 41776360›Full record

ReviewNpj biological timing and sleep2025

Temperature compensation of the Arabidopsis circadian clock.

Akari E Maeda, Norihito Nakamichi

Abstract readReview
In one paragraph

Review in Npj biological timing and sleep, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Intrinsic period stability of the cyanobacterial circadian oscillator across in vitro and in vivo conditions.Proceedings of the National Academy of Sciences of the United States of America · 2026
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Akari E MaedaGraduate School of Bioagricultural Sciences, Nagoya University, Furo-cho, Chikusa, Nagoya, Aichi, Japan.
Norihito NakamichiGraduate School of Bioagricultural Sciences, Nagoya University, Furo-cho, Chikusa, Nagoya, Aichi, Japan. nnaka@agr.nagoya-u.ac.jp.

Funding

Japan Society for the Promotion of Science 24KJ1268Ministry of Education, Culture, Sports, Science and Technology JP23H04197
6 · The paper itself

Abstract

The circadian clock is an endogenous, self-sustaining oscillator with a period of approximately 24 h. This timekeeping system helps organisms align their physiologies and behaviors with the day-night cycle. Temperature compensation, which allows the clock to maintain a period of about 24 h across physiological temperature ranges, is striking because, the rates of biochemical reaction rates increase with temperature in general. This review summarizes the effects of temperature on the period length of circadian rhythms in plants, especially Arabidopsis thaliana (Arabidopsis). Most core clock genes contribute to the temperature compensation of circadian period length. Temperature compensation involves the active, targeted degradation of specific core clock proteins at lower temperatures. Comparing the molecular mechanisms across different organisms may suggest design principles that allow evolutionarily distinct clock proteins to achieve temperature compensation.

Identifiers

PMID41776360
PMCPMC12912380

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.