Evidence map›Paper›PMID 41775896›Full record

ArticleNPJ cardiovascular health2025

Associations between carotid artery intima-media thickness, traditional risk factors and proteins.

Lars Lind, Rui Zheng

Abstract read
In one paragraph

Article in NPJ cardiovascular health, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Lars LindDepartment of Medical Sciences, Uppsala University, Uppsala, Sweden.
Rui ZhengDepartment of Medical Sciences, Uppsala University, Uppsala, Sweden. rui.zheng@uu.se.

Funding

Akademiska Sjukhuset not applicable
6 · The paper itself

Abstract

We aimed to assess the role of traditional cardiovascular risk factors on carotid artery intima-media thickness (IMT) and proteins associated with IMT. IMT was measured in 50,704 participants from the UK Biobank. Plasma levels of 2923 proteins were analyzed in 6328 individuals. Mendelian randomization (MR) analyses used genetic data on IMT, proteins, and risk factors. Observational analyses showed positive associations of LDL-cholesterol, body mass index (BMI), diabetes, and systolic blood pressure (SBP) with IMT, while HDL-cholesterol was inversely related. MR analysis confirmed causality for LDL, BMI, diabetes, but not HDL. Of 63 proteins observationally linked to IMT, 17 were replicated externally. Five proteins (BCAM, NTproBNP/NPPB, RABEPK, ACAN, FN1) were associated with IMT in MR, with RABEPK showing concordant directional relationships. MR supports causal links between LDL, BMI, diabetes, SBP, and IMT. RABEPK emerged as a potential therapeutic target, warranting further investigation.

Identifiers

PMID41775896
PMCPMC12912406

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.