In one paragraphArticle in The Journal of infectious diseases, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registryThe trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
3 · Its place in the literatureWho cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
4 · The recordCorrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what moneyAuthors and funding
10 authors.
Christian Rosas-SalazarDepartment of Pediatrics, Vanderbilt University Medical Center, Nashville, Tennessee, USA.ORCID 0000-0001-6644-5780 Tebeb GebretsadikDepartment of Biostatistics, Vanderbilt University Medical Center, Nashville, Tennessee, USA.
James D ChappellDepartment of Pediatrics, Vanderbilt University Medical Center, Nashville, Tennessee, USA.
R Stokes PeeblesDepartment of Medicine, Vanderbilt University Medical Center, Nashville, Tennessee, USA.
William D DupontDepartment of Biostatistics, Vanderbilt University Medical Center, Nashville, Tennessee, USA.ORCID 0000-0002-8166-599X Meghan H ShiltsDepartment of Medicine, Vanderbilt University Medical Center, Nashville, Tennessee, USA.ORCID 0000-0002-3879-6070 Samadhan J JadhaoDepartment of Pediatrics, Emory University School of Medicine and Children's Healthcare of Atlanta, Atlanta, Georgia, USA.
Larry J AndersonDepartment of Pediatrics, Emory University School of Medicine and Children's Healthcare of Atlanta, Atlanta, Georgia, USA.
Suman R DasDepartment of Medicine, Vanderbilt University Medical Center, Nashville, Tennessee, USA.
Tina V HartertDepartment of Pediatrics, Vanderbilt University Medical Center, Nashville, Tennessee, USA.ORCID 0000-0001-7470-1166 Funding
The Vanderbilt Institute for Clinical and Translational Research (VICTR)UL1TR000445 · NCATS · VANDERBILT UNIVERSITY MEDICAL CENTER · PI BERNARD, GORDON RAPHAEL · 2012 to 2016
$41.4MViral and Host Determinants of Infant and Childhood Allergy and AsthmaU19AI095227 · NIAID · VANDERBILT UNIVERSITY MEDICAL CENTER · PI Christian Rosas-Salazar · 2011 to 2026
$34.9MThe J. Craig Venter Institute Genome Center for Infectious DiseasesU19AI110819 · NIAID · J. CRAIG VENTER INSTITUTE, INC. · PI TAN, GENE · 2014 to 2018
$29.7MWisconsin Infant Study Cohort (WISC) ECHO Pediatric Follow-UpUG3OD023282 · OD · UNIVERSITY OF WISCONSIN-MADISON · PI GERN, JAMES E. · 2016 to 2024
$19.3MMidcareer Investigator Patient-oriented Research Award in Bronchiolitis & AsthmaK24AI077930 · NIAID · VANDERBILT UNIVERSITY MEDICAL CENTER · PI HARTERT, TINA V · 2008 to 2017
$1.6MThe effect of early-life nasal colonization with Lactobacillus on the development of the local immune response and the later onset of recurrent wheeze, childhood asthma, and allergic rhinitisK23HL148638 · NHLBI · VANDERBILT UNIVERSITY MEDICAL CENTER · PI ROSAS-SALAZAR, CHRISTIAN · 2019 to 2023
$909kNational Institute of Allergy and Infectious Diseases K24AI77930National Institute of Allergy and Infectious Diseases U19AI095227National Institute of Allergy and Infectious Diseases U19AI110819National Institute of Allergy and Infectious Diseases UG3OD023282NCATS NIH HHS UL1 TR000445NCATS NIH HHS UL1TR000445NHLBI NIH HHS K23 HL148638NHLBI NIH HHS K23HL148638NIAID NIH HHS K24 AI077930NIAID NIH HHS U19 AI095227NIAID NIH HHS U19 AI110819NIHNIH HHS UG3 OD023282Vanderbilt Institute for Clinical and Translational Research
6 · The paper itselfAbstract
Our objective was to test the hypothesis that respiratory syncytial virus (RSV) infection during infancy with genotypes containing previously reported G gene sequence duplications (Gdups) is associated with an increased risk of childhood asthma. In a population-based, prospective, birth cohort of healthy, term children, we found that children with RSV-A Gdup+ (adjusted odds ratio [aOR] = 2.00, 95%CI = 1.15-3.47) and RSV-B Gdup + (aOR = 1.78, 95%CI = 1.01-3.11) infections during infancy had higher odds of 5-year current asthma than those without RSV infection during infancy. These findings are proof-of-concept evidence that RSV genetic variation is associated with variable childhood asthma risk.
Indexed as
AsthmaRespiratory Syncytial Virus, HumanRespiratory Syncytial Virus InfectionsBirth CohortChild, PreschoolFemaleGenotypeHumansInfantMaleProspective StudiesRisk FactorsasthmachildrengenotypeG generespiratory syncytial virus
Identifiers
PMID41773827
PMCPMC13431657
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