ArticleCureus2026
Acute Renal Infarction Secondary to Atrial Fibrillation: A Rare Cause of Sudden Flank Pain.
Article in Cureus, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
5 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Acute renal infarction is an uncommon and often underrecognized cause of sudden flank pain, posing diagnostic challenges due to its nonspecific presentation and laboratory findings. It may manifest with abrupt, severe flank discomfort, nausea, vomiting, and microscopic hematuria, frequently mimicking more common renal or musculoskeletal conditions. Risk factors such as atrial fibrillation significantly increase the likelihood of embolic events leading to segmental renal ischemia, particularly in patients with suboptimal anticoagulation. Imaging, especially contrast-enhanced computed tomography, is pivotal in establishing the diagnosis by identifying characteristic wedge-shaped perfusion defects, while supportive laboratory markers, including elevated lactate dehydrogenase and leukocytosis, may provide additional clues. Prompt recognition and initiation of anticoagulation therapy are central to preserving renal function and preventing further embolic complications. Clinical vigilance, timely diagnostic evaluation, and careful management can result in favorable outcomes, even in patients presenting with acute kidney injury, highlighting the importance of considering renal infarction in the differential diagnosis of sudden flank pain, particularly in those with cardiovascular risk factors.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.