ArticleThe Journal of cell biology2026
Two δ-catenins, plakophilin 4 and p120, promote formation of distinct types of adherens junctions.
Article in The Journal of cell biology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
Classical cadherins are instrumental for connecting cells into tissues by forming adherens junctions (AJs), a structurally diverse class of cell-cell adhesions tailored to specific membrane domains, cell types, and particular functions. The mechanisms that underlie the AJ diversification remain unknown. Here, we show that two δ-catenin family members, p120 and plakophilin 4 (pkp4), which bind the intracellular region of classical cadherins, promote distinct modes of cadherin clustering, thereby contributing to AJ specialization. The mode promoted by p120 is driven by interactions between cadherin-associated protein, α-catenin, and actin filaments. This "canonical" clustering mechanism generates apical and basal AJs that play a major role in overall cell-cell adhesion. The mode promoted by pkp4 is driven by an α-catenin-independent mechanism. It generates lateral AJs, which apparently function in processes other than cell-cell adhesion. Collectively, our findings show that δ-catenins regulate the balance between different AJ assembly pathways, thereby contributing to AJ diversification.
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