Evidence map›Paper›PMID 41727076›Full record

ArticlebioRxiv : the preprint server for biology2026

AAV-only targeting of ventral tegmental area dopamine neurons for optical self-stimulation studies in mice.

Ezequiel Marron Fernandez de Velasco, John C Brent, Alex L Welter, Praseuth Yang, Annelise Wickman, Eric H Mitten, Kevin Wickman

Abstract readPreprint
In one paragraph

Article in bioRxiv : the preprint server for biology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

7 authors.

Ezequiel Marron Fernandez de VelascoDepartment of Pharmacology University of Minnesota Minneapolis, MN USA 55455.
John C BrentDepartment of Pharmacology University of Minnesota Minneapolis, MN USA 55455.
Alex L WelterMolecular Pharmacology and Therapeutics Graduate Program University of Minnesota Minneapolis, MN USA 55455.
Praseuth YangDepartment of Pharmacology University of Minnesota Minneapolis, MN USA 55455.
Annelise WickmanDepartment of Pharmacology University of Minnesota Minneapolis, MN USA 55455.
Eric H MittenGraduate Program in Neuroscience University of Minnesota Minneapolis, MN USA 55455.
Kevin WickmanDepartment of Pharmacology University of Minnesota Minneapolis, MN USA 55455.ORCID 0000-0002-5179-9540

Funding

Viral Innovation CoreP30DA048742 · NIDA · UNIVERSITY OF MINNESOTA · PI Anna Zilverstand · 2020 to 2026
$19.0M
NEUROSCIENCE TRAINING IN DRUG ABUSE RESEARCHT32DA007234 · NIDA · UNIVERSITY OF MINNESOTA TWIN CITIES · PI Paul G Mermelstein, Jocelyn M Richard · 1986 to 2026
$10.9M
Predoctoral Training of NeuroscientistsT32NS105604 · NINDS · UNIVERSITY OF MINNESOTA · PI A DAVID REDISH · 2018 to 2026
$3.5M
Relevance and plasticity of inhibitory metabotropic signaling in reward circuitsR01DA034696 · NIDA · UNIVERSITY OF MINNESOTA · PI WICKMAN, KEVIN D · 2013 to 2022
$3.4M
Alcohol-related suppression of GIRK channel activity in the basal amygdala: a link to plasticity of glutamatergic neurotransmission and withdrawal-associated behavior?R01AA027544 · NIAAA · UNIVERSITY OF MINNESOTA · PI WICKMAN, KEVIN D · 2020 to 2024
$1.7M
Minnesota Inclusive Neuroscience Development Scholars (MINDS) doctoral readiness programR25DA057802 · NIDA · UNIVERSITY OF MINNESOTA · PI Angeline Joan Dukes, Julia C Lemos · 2022 to 2026
$1.4M
Elucidating the Mechanisms and Relevance of Cocaine-Induced Plasticity of Inhibitory G Protein Signaling in the Prelimbic CortexF31DA062412 · NIDA · UNIVERSITY OF MINNESOTA · PI Alex Larsen Welter · 2025 to 2026
$95k
NIAAA NIH HHS R01 AA027544NIDA NIH HHS F31 DA062412NIDA NIH HHS P30 DA048742NIDA NIH HHS R01 DA034696NIDA NIH HHS R25 DA057802NIDA NIH HHS T32 DA007234NINDS NIH HHS T32 NS105604
6 · The paper itself

Abstract

Studies employing optogenetic approaches in rodent models have highlighted the important contribution of ventral tegmental area (VTA) dopamine (DA) neurons to reward, learning, and motivation. Selective manipulation of VTA DA neurons is generally achieved in these studies using transgenic mouse or rat lines that express Cre recombinase under the control of a promoter active in DA neurons, combined with intra-VTA infusion of adeno-associated virus (AAV) vectors harboring Cre recombinase-dependent expression cassettes. Reliance on transgenic Cre driver lines is expensive and decreases study efficiency, and available driver lines have unique limitations. Here, we report the development of an AAV-only approach that permits genetic access to VTA DA neurons and can support optogenetic self-stimulation in mice. We used a 2.5 kb fragment of the mouse tyrosine hydroxylase promoter (mTH) to drive Cre expression in VTA DA neurons. Intra-VTA co-infusion of AAV8-mTH-Cre with an AAV vector harboring a Cre-dependent yellow fluorescent protein expression cassette yielded high efficiency (82%) and high fidelity (73%) targeting of tyrosine hydroxylase-positive VTA neurons in C57BL/6J mice. Co-infusion of AAV8-mTH-Cre with a vector harboring a Cre-dependent channelrhodopsin (ChR2) expression cassette permitted optical regulation of VTA neurons with electrophysiological features consistent with VTA DA neurons. Moreover, C57BL/6J mice expressing ChR2 in VTA DA neurons rapidly acquired optical self-stimulation behavior. Thus, this AAV-only approach should facilitate investigation of VTA DA neuron contributions to reward-related behaviors and permit comparative assessments in reward circuit function in inbred and mutant mouse strains.

Indexed as

adeno-associated vectordopamine neuronmiceoptogeneticself-stimulationventral tegmental area

Identifiers

PMID41727076
PMCPMC12919011

What OpenQuestion holds

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LicenceCC BY-NC-ND
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.