ReviewSub-cellular biochemistry2026
Golgi Drivers of Cancer.
Review in Sub-cellular biochemistry, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
1 citing paper in PubMed.
- Oncogenic receptor tyrosine kinase signaling is driven by the Golgi protein GOLPH3 and its interaction with MYO18A.Science signaling · 2026Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
4 authors.
Funding
Abstract
Unbiased analyses of cancer genomes, transcriptomes, and proteomes have led to the identification of numerous genes that, when perturbed, contribute to the formation of cancer in humans. These unbiased analyses, by identifying drivers of cancer that localize to and function at the Golgi, have illuminated a previously unappreciated role for the Golgi in driving oncogenic phenotypes. Here we review some of the cancer drivers that act at the Golgi. We highlight the evidence that they are functionally important in cancer, and the evidence that they localize to and function at the Golgi. We also discuss functional interactions between these proteins, and review some of the proposed mechanisms by which they promote oncogenic phenotypes.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.