Evidence map›Paper›PMID 41714800›Full record

ArticleCommunications biology2026

Dendritic heterosynaptic plasticity arises from calcium-based input learning.

Shirin Shafiee, Sebastian Schmitt, Christian Tetzlaff

Abstract read
In one paragraph

Article in Communications biology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Article
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors.

Shirin ShafieeIII. Institute of Physics-Biophysics, Faculty of Physics, University of Göttingen, Göttingen, Germany. shirin.shafieekamalabad@uni-goettingen.de.ORCID http://orcid.org/0009-0009-7271-8939
Sebastian SchmittIII. Institute of Physics-Biophysics, Faculty of Physics, University of Göttingen, Göttingen, Germany.ORCID http://orcid.org/0000-0002-7935-0470
Christian TetzlaffIII. Institute of Physics-Biophysics, Faculty of Physics, University of Göttingen, Göttingen, Germany.ORCID http://orcid.org/0000-0003-1901-6232

Funding

Deutsche Forschungsgemeinschaft (German Research Foundation) SFB1286/C01&Z01, TE 1172/7-1
6 · The paper itself

Abstract

Stimulus-triggered synaptic plasticity is the foundation of learning and crucial cognitive abilities. Although numerous computational models have investigated plasticity within networks of point neurons, dendritic integration confers superior computational capacity compared to these simplistic models, highlighting the significance of dendrites and their spines-small, specialized protrusions that serve as loci for synaptic plasticity. Synaptic plasticity can be categorized into two forms: homosynaptic plasticity, involving changes at directly stimulated synapses, and heterosynaptic plasticity, involving changes at non-stimulated synapses. For homosynaptic plasticity, the Ca

Indexed as

CalciumDendritesLearningNeuronal PlasticitySynapsesAnimalsCalcium SignalingDendritic SpinesModels, NeurologicalCalcium

Identifiers

PMID41714800
PMCPMC12993064

What OpenQuestion holds

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LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.