Evidence map›Paper›PMID 41704835›Full record

ArticleEuropean heart journal. Case reports2026

Bidirectional ventricular tachycardia caused by occlusion myocardial infarction-a case report.

Magnus Nossen, Mathis Korseberg Stokke

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Article in European heart journal. Case reports, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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1 · What the graph read from it

What it found

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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

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3 · Its place in the literature

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0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

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5 · Who and what money

Authors and funding

2 authors.

Magnus NossenInstitute for Experimental Medical Research, Oslo University Hospital, Ullevål Kirkeveien 166, Oslo 0450, Norway.ORCID https://orcid.org/0009-0003-1571-1171
Mathis Korseberg StokkeInstitute for Experimental Medical Research, Oslo University Hospital, Ullevål Kirkeveien 166, Oslo 0450, Norway.ORCID https://orcid.org/0000-0002-8869-8153

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Background: Bidirectional ventricular tachycardia (BdVT) is an uncommon form of ventricular arrhythmia typically associated with digoxin toxicity or catecholaminergic polymorphic ventricular tachycardia. BdVT in the setting of acute myocardial ischaemia is limited to case reports. We present an example of BdVT caused by occlusion myocardial infarction with original and modified Sgarbossa criteria being positive. Case summary: An elderly male with known coronary artery disease presented with chest pain. The initial ECG demonstrated a regular wide QRS complex tachycardia with alternating frontal plane axis, consistent with BdVT. No apparent cause of the arrhythmia was identified from the patient's medication history or family history. Detailed ECG analysis revealed that during tachycardia, the QRS complexes exhibited excessively discordant ST segment elevation and depression. ST segment concordance was observed in natively conducted beats of left bundle branch block morphology following termination of the arrhythmia. These findings raised suspicion of occlusion myocardial infarction as the direct cause of BdVT. This was subsequently confirmed by coronary angiography. Conclusion: BdVT is a distinct form of ventricular tachycardia, characterized by beat-to-beat alternation in QRS axis and morphology. BdVT in the context of acute ischaemia is rare and limited to case reports. Although this clinical presentation is exceptionally rare, in patients with BdVT and symptoms suggestive of acute coronary syndrome, active myocardial ischaemia should be considered as a potential underlying cause. This case highlights the utility of both the original and modified Sgarbossa criteria in identifying acute ischaemia in the setting of a wide complex rhythm.

Indexed as

Acute coronary syndromeArrhythmiaBidirectional ventricular tachycardiaCase reportOcclusion myocardial infarctionSgarbossa criteria

Identifiers

PMID41704835
PMCPMC12908186

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