ArticleWorld journal of microbiology & biotechnology2026
Astaxanthin biosynthesis stimulation in wild type Xanthophyllomyces dendrorhous allows transcriptional reprogramming of genetic responses involved in oxidative stress and mitochondrial defense.
Article in World journal of microbiology & biotechnology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
Astaxanthin is an attractive carotenoid that contains beneficial properties as a powerful antioxidant capable of preventing and treating degenerative diseases, while Xanthophyllomyces dendrorhous is one of its main sources. Astaxanthin from X. dendrorhous is generated in response to oxidative or metabolic stress but, to increase the astaxanthin yields, it is necessary to stimulate the metabolic pathway in a way that allows the transcriptional reprogramming of involved genes. Research about the stimulation of carotenoids metabolic pathway posed an important question, could the expression of genes involved in the oxidative stress response be reprogrammed to enhance astaxanthin biosynthesis? Several investigations have demonstrated that astaxanthin biosynthesis is assisted and regulated by electron donor proteins similar to cytochrome P450 reductase (encoded by crtR). Here, we assessed some of these reprogrammed transcriptional responses in wild-type X. dendrorhous under two stimulating conditions: (i) abscisic acid (ABA), and (ii) 6-benzylaminopurine (6-BAP) combined with H
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