Evidence map›Paper›PMID 41677628›Full record

ReviewCells2026

Molecular and Cellular Mechanisms of Myocardial Ischemia and Reperfusion Injury: A Narrative Review.

Stefan Juricic, Jovana Klac, Sinisa Stojkovic, Branko Beleslin, Milorad Tesic, Ivana Jovanovic, Marko Banovic, Olga Petrovic, Srdjan Aleksandric, Natalija Vasic and 5 more

Abstract readReview
In one paragraph

Review in Cells, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 12 papers.

0numbers the graph read from it
0cells of the map it votes in
12citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

12 citing papers in PubMed.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

15 authors.

Stefan JuricicClinic for Cardiology, University Clinical Center of Serbia, 11000 Belgrade, Serbia.ORCID 0000-0002-3045-5263
Jovana KlacClinic for Cardiology, University Clinical Center of Serbia, 11000 Belgrade, Serbia.
Sinisa StojkovicClinic for Cardiology, University Clinical Center of Serbia, 11000 Belgrade, Serbia.ORCID 0000-0002-5784-9377
Branko BeleslinClinic for Cardiology, University Clinical Center of Serbia, 11000 Belgrade, Serbia.ORCID 0000-0001-9452-8241
Milorad TesicClinic for Cardiology, University Clinical Center of Serbia, 11000 Belgrade, Serbia.ORCID 0000-0002-3758-3719
Ivana JovanovicClinic for Cardiology, University Clinical Center of Serbia, 11000 Belgrade, Serbia.
Marko BanovicClinic for Cardiology, University Clinical Center of Serbia, 11000 Belgrade, Serbia.ORCID 0000-0002-0017-6140
Olga PetrovicClinic for Cardiology, University Clinical Center of Serbia, 11000 Belgrade, Serbia.ORCID 0000-0002-6631-6353
Srdjan AleksandricClinic for Cardiology, University Clinical Center of Serbia, 11000 Belgrade, Serbia.ORCID 0000-0001-6903-4477
Natalija VasicDepartment of Cardiology, Emergency Center, University Clinical Center of Serbia, 11000 Belgrade, Serbia.
Filip SimeunovicFaculty of Medicine, University of Belgrade, 11000 Belgrade, Serbia.
Dejan LazovicFaculty of Medicine, University of Belgrade, 11000 Belgrade, Serbia.
Milica StoiljkovicFaculty of Medicine, University of Belgrade, 11000 Belgrade, Serbia.
Sashko NikolovFaculty of Medical Sciences, Goce Delcev University, 2000 Stip, North Macedonia.
Dejan SimeunovicClinic for Cardiology, University Clinical Center of Serbia, 11000 Belgrade, Serbia.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Myocardial ischemia represents a state of reduced coronary perfusion with oxygenated blood, insufficient to meet the metabolic demands of the myocardium. Both acute and chronic ischemia trigger a cascade of cellular events that lead to disturbances in ionic balance, mitochondrial function and energy metabolism. During ischemia, cardiomyocytes (CMs) shift from aerobic to anaerobic metabolism, resulting in adenosine triphosphate (ATP) depletion, loss of ionic homeostasis and calcium (Ca

Indexed as

Myocardial IschemiaMyocardial Reperfusion InjuryAnimalsApoptosisHumansMyocytes, CardiacOxidative StressReactive Oxygen SpeciesReactive Oxygen Speciesapoptosismitochondrial dysfunctionmyocardial ischemianecroptosisreperfusion injuryROS

Identifiers

PMID41677628
PMCPMC12896409

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.