Evidence map›Paper›PMID 41675617›Full record

ArticleCurrent research in microbial sciences2026

Loss of cPLA2α function attenuates inflammation and epithelial thickening in a mouse model of

Brice Lagrange, Fatima Benmohamed, Alice Eon-Bertho, Laurette Malleret, Alexis Trecourt, Olivier Glehen, Lhousseine Touqui, Abderrazzak Bentaher

Abstract read
In one paragraph

Article in Current research in microbial sciences, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Brice LagrangeEquipe, Inflammation et Immunité de l'Epithélium Respiratoire, INSERM, France.
Fatima BenmohamedSorbonne Université, INSERM UMR-S938, Centre de Recherche Saint-Antoine (CRSA), Paris, France.
Alice Eon-BerthoSorbonne Université, INSERM UMR-S938, Centre de Recherche Saint-Antoine (CRSA), Paris, France.
Laurette MalleretEquipe, Inflammation et Immunité de l'Epithélium Respiratoire, INSERM, France.
Alexis TrecourtUR 3738 CICLY, Faculté de Médecine Lyon Sud, France.
Olivier GlehenUR 3738 CICLY, Faculté de Médecine Lyon Sud, France.
Lhousseine TouquiSorbonne Université, INSERM UMR-S938, Centre de Recherche Saint-Antoine (CRSA), Paris, France.
Abderrazzak BentaherEquipe, Inflammation et Immunité de l'Epithélium Respiratoire, INSERM, France.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Among the characteristics of chronic obstructive pulmonary diseases (COPD) especially emphysema are chronic airway inflammation and tissue destruction in response to the primary etiologic factor, cigarette smoking (CS). Importantly, COPD is aggravated by respiratory infections. Approximately half of these exacerbations are caused by bacterial infections with

Indexed as

COPD exacerbationCytosolic phospholipase 2αHaemophilus influenzaelung inflammationLung injury

Identifiers

PMID41675617
PMCPMC12887665

What OpenQuestion holds

Textmetadata
LicenceCC BY
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.