Evidence map›Paper›PMID 41623903›Full record

ArticleJournal of the Endocrine Society2026

Molecular Genetics of 1α-Hydroxylase Deficiency in the Saudi Population.

Bassam Bin-Abbas, Afaf Alsagheir, Balgees Alghamdi, Allianah Benito, Yufei Shi, Somaya Khader Alzelaye, Noman Ahmad, Fahad Al Juraibah, Amer Omar Alali, Adnan Al Shaikh and 7 more

Abstract read
In one paragraph

Article in Journal of the Endocrine Society, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

17 authors.

Bassam Bin-AbbasSection of Pediatric Endocrinology, Department of Pediatrics, King Faisal Specialist Hospital & Research Centre, Riyadh 11211, Saudi Arabia.
Afaf AlsagheirSection of Pediatric Endocrinology, Department of Pediatrics, King Faisal Specialist Hospital & Research Centre, Riyadh 11211, Saudi Arabia.ORCID https://orcid.org/0000-0002-1844-4377
Balgees AlghamdiDepartment of Molecular Oncology, Research and Innovation, King Faisal Specialist Hospital and Research Centre, Riyadh 11211, Saudi Arabia.
Allianah BenitoDepartment of Molecular Oncology, Research and Innovation, King Faisal Specialist Hospital and Research Centre, Riyadh 11211, Saudi Arabia.
Yufei ShiDepartment of Molecular Oncology, Research and Innovation, King Faisal Specialist Hospital and Research Centre, Riyadh 11211, Saudi Arabia.ORCID https://orcid.org/0000-0002-6999-0191
Somaya Khader AlzelayeCentre of Endocrinology and Diabetes Mellitus, Al-Qunfudah General Hospital, Al-Qunfudah 28814, Saudi Arabia.
Noman AhmadDepartment of Pediatrics, King Faisal Specialist Hospital and Research Centre, Jeddah 23433, Saudi Arabia.
Fahad Al JuraibahCollege of Medicine, King Saud bin Abdul-Aziz University for Health Sciences, Ministry of National Guard Health Affairs, Riyadh 11481, Saudi Arabia.
Amer Omar AlaliDepartment of Pediatrics, Jazan Hospital, Jazan 82874, Saudi Arabia.
Adnan Al ShaikhDepartment of Pediatrics, King Abdulaziz Medical City (KAMC), King Saud Bin Abdulaziz University for Health Sciences (KSAU-HS), King Abdullah International Medical Research Centre (KAIMRC), Ministry of National Guard Health Affairs (MNGHA), Jeddah 21423, Saudi Arabia.
Najya AttiaDepartment of Pediatrics, King Abdulaziz Medical City (KAMC), King Saud Bin Abdulaziz University for Health Sciences (KSAU-HS), King Abdullah International Medical Research Centre (KAIMRC), Ministry of National Guard Health Affairs (MNGHA), Jeddah 21423, Saudi Arabia.
Abdulhameed Abdulmohsen AlbunyanDepartment of Paediatrics, MOH-Saudi Arabia, Maternity and Children Hospital Alhasa-Hofuf 36422, Saudi Arabia.
Abdullah Saad AlshahranyDepartment of Pediatrics, Armed Forces Hospital Southern Region, Khamyes Mushait 62413, Saudi Arabia.
Ahmed Ali NahariDepartment of Pediatrics, Jazan Specialist Hospital, Jazan 45142, Saudi Arabia.
Nabilah SulaimaniDepartment of Pediatrics,Maternity and Children Hospital-Makkah, Makkah 24246, Saudi Arabia.
Khloud M AlrubayaDepartment of Pediatrics, Maternity and Children Hospital-Dammam, Dammam 32253, Saudi Arabia.
Ali S AlzahraniDepartment of Molecular Oncology, Research and Innovation, King Faisal Specialist Hospital and Research Centre, Riyadh 11211, Saudi Arabia.ORCID https://orcid.org/0000-0003-4294-3624

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Context: The aim of this study was to characterize the molecular genetics of 1α-hydroxylase deficiency in the highly consanguineous population of Saudi Arabia, hypothesizing that the results will show a unique Methods: We collected data on a large cohort of patients diagnosed with 1α-hydroxylase deficiency from different parts of the country. These patients underwent molecular testing for Results: A cohort of 45 patients from 29 unrelated families was studied. In 13 families (29 patients), more than one affected sibling was included (2-4 siblings) while the other 16 families had only a single patient per family. The patients included 24 females and 21 males with median age at time of presentation of 1 year and a current median age of 10 years. The clinical, biochemical and radiological profile was typical of 1α-hydroxylase deficiency. Molecular testing showed 10 mutations of different types in the 29 families. Four mutations were novel (p.(Trp257LeufsTer76), p.(Glu101Gln), p.(Gly398Ser), and p.(Arg206Cys)) while the other 6 mutations were previously described (p.(arg429Pro), p.(Phe443Profs*24), p.(Gln135Ter), p. (Gly102Glu), p.(Gln504Ter), and c.589 + 1G > A). Two of the previously reported mutations were from Saudi patients and have never been reported from other populations, increasing the number of novel/previously novel mutations to 6 of 10 mutations (60%). The most common mutation was c.1286G > C, p.(Arg429Pro) occurring in 22 patients from 15 unrelated families (51.7%). Conclusion: The molecular genetics of 1α-hydroxylase deficiency in Saudi Arabia is unique with several novel mutations of different types and a possible founder mutation.

Indexed as

1α-hydroxylaseCYP27B1vitamin D–dependent ricketsvitamin D–resistant rickets type 1A

Identifiers

PMID41623903
PMCPMC12852950

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.