Evidence map›Paper›PMID 41613680›Full record

ArticleCureus2025

Age-Related Changes in Matrix Metalloproteinase-9 Expression in Spinal Motor Neurons of Normal Mice.

Jun Yamazaki, Takuto Hideyama, Sayaka Teramoto, Haruhisa Kato, Hitoshi Aizawa, Shin Kwak, Hiroo Terashi

Abstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Cureus, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Jun YamazakiNeurology, Tokyo Medical University Hospital, Tokyo, JPN.
Takuto HideyamaNeurology, Tokyo Medical University Hospital, Tokyo, JPN.
Sayaka TeramotoNeurology, Tokyo Medical University Hospital, Tokyo, JPN.
Haruhisa KatoNeurology, Tokyo Medical University Hospital, Tokyo, JPN.
Hitoshi AizawaNeurology, Tokyo Medical University Hospital, Tokyo, JPN.
Shin KwakNeurology, Tokyo Medical University Hospital, Tokyo, JPN.
Hiroo TerashiNeurology, Tokyo Medical University Hospital, Tokyo, JPN.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disorder involving the degeneration of upper motor neurons (UMNs) and lower motor neurons (LMNs). Although the cause of motor neuron (MN) degeneration in patients with ALS remains unknown, certain MN types (such as oculomotor neurons) and MNs within the Onuf (Onuf-Mannen) nucleus are preserved until the terminal stage. We previously generated mice with a selective knockout of adenosine deaminase acting on RNA 2 (ADAR2) in cholinergic neurons (

Indexed as

adenosine deaminase acting on rna 2 (adar2)amyotrophic lateral sclerosis (als)fast-fatigable motor neurons (ff mns)matrix metalloproteinase-9 (mmp-9)tar dna-binding protein 43 (tdp-43)

Identifiers

PMID41613680
PMCPMC12848547

What OpenQuestion holds

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LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.