Evidence map›Paper›PMID 41611798›Full record

ArticleScientific reports2026

Heritable pulmonary arterial hypertension: new genetic findings and environmental triggers.

Memoona Shaukat, Ekkehard Grünig, Simon Haas, Jan Haas, Mohammad Panahi, Martin Granzow, Tobias J Lange, Stefan Stadler, Natascha Sommer, Peter Dorfmüller and 6 more

Abstract read
In one paragraph

Article in Scientific reports, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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0cells of the map it votes in
0citing papers in PubMed
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1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

16 authors.

Memoona ShaukatCenter for Pulmonary Hypertension, Thoraxklinik Heidelberg gGmbH at Heidelberg University Hospital and Translational Lung Research Center Heidelberg (TLRC), German Center for Lung Research (DZL), Heidelberg, Germany.
Ekkehard GrünigCenter for Pulmonary Hypertension, Thoraxklinik Heidelberg gGmbH at Heidelberg University Hospital and Translational Lung Research Center Heidelberg (TLRC), German Center for Lung Research (DZL), Heidelberg, Germany.
Simon HaasCenter for Pulmonary Hypertension, Thoraxklinik Heidelberg gGmbH at Heidelberg University Hospital and Translational Lung Research Center Heidelberg (TLRC), German Center for Lung Research (DZL), Heidelberg, Germany.
Jan HaasInstitute for Cardiomyopathies Heidelberg (ICH) and Center for Cardiogenetics, Heidelberg University Hospital, and DZHK, Site Heidelberg/Mannheim, Heidelberg, Germany.
Mohammad PanahiCenter for Pulmonary Hypertension, Thoraxklinik Heidelberg gGmbH at Heidelberg University Hospital and Translational Lung Research Center Heidelberg (TLRC), German Center for Lung Research (DZL), Heidelberg, Germany.
Martin GranzowLaboratory for Molecular Genetic Diagnostics, Institute of Human Genetics, Heidelberg University, Heidelberg, Germany.
Tobias J LangeDepartment of Pneumology, Kliniken Südostbayern AG, Kreisklinik Bad Reichenhall, Bad Reichenhall, Germany.
Stefan StadlerDepartment of Internal Medicine II, University Medical Center Regensburg, Regensburg, Germany.
Natascha SommerMedical and Policlinic II, Excellence Cluster Cardio-Pulmonary Institute (CPI), Member of the German Center for Lung Research (DZL), Justus-Liebig University Giessen, Giessen, Germany.
Peter DorfmüllerInstitute for Lung Health (ILH), Giessen, University Hospital of Gießen and Marburg, Universities of Giessen and Marburg Lung Center (UGMLC), German Center for Lung Research (DZL), Gießen, Germany.
Benjamin MederInstitute for Cardiomyopathies Heidelberg (ICH) and Center for Cardiogenetics, Heidelberg University Hospital, and DZHK, Site Heidelberg/Mannheim, Heidelberg, Germany.
Satenik HarutyunovaCenter for Pulmonary Hypertension, Thoraxklinik Heidelberg gGmbH at Heidelberg University Hospital and Translational Lung Research Center Heidelberg (TLRC), German Center for Lung Research (DZL), Heidelberg, Germany.
Benjamin EgenlaufCenter for Pulmonary Hypertension, Thoraxklinik Heidelberg gGmbH at Heidelberg University Hospital and Translational Lung Research Center Heidelberg (TLRC), German Center for Lung Research (DZL), Heidelberg, Germany.
Panagiota XanthouliCenter for Pulmonary Hypertension, Thoraxklinik Heidelberg gGmbH at Heidelberg University Hospital and Translational Lung Research Center Heidelberg (TLRC), German Center for Lung Research (DZL), Heidelberg, Germany.
Katrin HinderhoferLaboratory for Molecular Genetic Diagnostics, Institute of Human Genetics, Heidelberg University, Heidelberg, Germany.
Christina A EichstaedtCenter for Pulmonary Hypertension, Thoraxklinik Heidelberg gGmbH at Heidelberg University Hospital and Translational Lung Research Center Heidelberg (TLRC), German Center for Lung Research (DZL), Heidelberg, Germany. christina.eichstaedt@med.uni-heidelberg.de.ORCID http://orcid.org/0000-0001-7288-8297

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Our goal was to identify new environmental or genetic causes in heritable pulmonary arterial hypertension (HPAH) families outside the 18 known diagnostics PAH genes. PAH gene panel sequencing was performed for 47 HPAH families which revealed pathogenic variants in 39 families. Five of the remaining families agreed to whole exome sequencing and to fill in a drug and toxin exposure questionnaire. In Family 1 and 2, mother and daughter with HPAH carried a likely pathogenic variant in the CYBA gene and a variant of uncertain significance in the FKBP1A gene, respectively, following ACMG guidelines. In Family 3, we detected a likely pathogenic variant in the PTGR2 gene. These genes could play part in PAH pathogenesis but further functional analyses are required to corroborate these findings. In the remaining two families, we could not identify any plausible genetic cause. However, a father and son with PAH reported exposure to trichloroethylene, asbestos and tramadol in Family 4. In Family 5, two brothers with pulmonary veno-occlusive disease showed occupational toxin exposure. Thus, our findings indicate that not only a genetic predisposition but also environmental triggers should be investigated for HPAH patients.

Indexed as

Environmental ExposureFamilial Primary Pulmonary HypertensionGenetic Predisposition to DiseaseHypertension, PulmonaryPulmonary Arterial HypertensionAdultExome SequencingFemaleHumansMaleMiddle AgedPedigreeDrug and toxin exposuresPulmonary arterial hypertensionTransforming growth factor betaWhole exome sequencing

Identifiers

PMID41611798
PMCPMC12858969

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.