Evidence map›Paper›PMID 41589112›Full record

ArticleJBMR plus2026

A reproducible mouse model of TMJ anterior disc displacement.

Maria Pacheco-Vergara, Daniela Gioia, Sebastian Ko, Naomi S Sta Maria, Francesca V Mariani, Amy E Merrill

Abstract read
In one paragraph

Article in JBMR plus, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Maria Pacheco-VergaraCenter for Craniofacial Molecular Biology, Department of Biomedical Sciences, Herman Ostrow School of Dentistry, University of Southern California, Los Angeles, CA 90033, United States.
Daniela GioiaDepartment of Stem Cell Biology and Regenerative Medicine, Keck School of Medicine, University of Southern California, Los Angeles, CA 90033, United States.
Sebastian KoCenter for Craniofacial Molecular Biology, Department of Biomedical Sciences, Herman Ostrow School of Dentistry, University of Southern California, Los Angeles, CA 90033, United States.
Naomi S Sta MariaZilkha Neurogenetic Institute, Keck School of Medicine, University of Southern California, Los Angeles, CA 90033, United States.
Francesca V MarianiDepartment of Stem Cell Biology and Regenerative Medicine, Keck School of Medicine, University of Southern California, Los Angeles, CA 90033, United States.ORCID https://orcid.org/0000-0003-1619-8763
Amy E MerrillCenter for Craniofacial Molecular Biology, Department of Biomedical Sciences, Herman Ostrow School of Dentistry, University of Southern California, Los Angeles, CA 90033, United States.ORCID https://orcid.org/0000-0002-3660-1575

Funding

THE ROLE OF FGFR2 IN PROTEIN SYNTHESIS DURING SKELETAL DEVELOPMENTR01DE025222 · NIDCR · UNIVERSITY OF SOUTHERN CALIFORNIA · PI MERRILL, AMY E · 2015 to 2024
$3.7M
Regulating Skeletal Connectivity in Craniofacial Development and DiseaseR35DE034346 · NIDCR · UNIVERSITY OF SOUTHERN CALIFORNIA · PI Amy E Merrill · 2024 to 2026
$2.3M
Developmental regulation of tendon-bone connectivity in the jawR01DE029850 · NIDCR · UNIVERSITY OF SOUTHERN CALIFORNIA · PI MERRILL, AMY E · 2021 to 2024
$1.8M
Cellular and molecular coordinators of large-scale bone repairR01AR083445 · NIAMS · UNIVERSITY OF SOUTHERN CALIFORNIA · PI Francesca V Mariani · 2024 to 2026
$1.7M
NIAMS NIH HHS R01 AR083445NIDCR NIH HHS R01 DE025222NIDCR NIH HHS R01 DE029850NIDCR NIH HHS R35 DE034346
6 · The paper itself

Abstract

Temporomandibular joint disorders (TMDs) are a group of clinical conditions characterized by chronic jaw dysfunction and persistent orofacial pain, affecting approximately one-third of the adult population. Anterior displacement of the articular disc (ADD), which leads to joint instability and mechanical overload, is strongly linked to arthrogenous TMD. However, the cellular and molecular mechanisms triggered by ADD, and how they contribute to joint derangement, remain poorly understood. Clinical treatments for this chronic condition are also limited. Although several animal models exist for ADD, most are difficult to validate and rely on species that lack advanced genetic tools. To address this, we developed a reproducible and compatible surgical murine model of ADD, which is suitable for lineage tracing, reporter analyses, and gene modification techniques. Here, we describe a procedure for generating unilateral surgical ADD in adult mice and validate this model using MRI. Whole-mount skeletal staining and histology reveal degeneration of the condylar cartilage, erosion of the temporal bone, and disc fibrosis by 15 d post-surgery (dps), compared to controls. Overall, this genetically tractable murine model offers a powerful platform for dissecting the mechanisms of ADD and identifying new cellular and molecular targets for TMD therapy.

Indexed as

animal modelanterior disc displacementarticular disccraniofacialjawjoint injury modelMRIsynovial jointTMDTMJ

Identifiers

PMID41589112
PMCPMC12832048

What OpenQuestion holds

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LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.