ArticleVeterinary research2026
Outer membrane vesicles secreted by avian pathogenic Escherichia coli promote its survival within macrophages and systemic infection by inducing endoplasmic reticulum stress-mediated autophagy flux blockade.
Article in Veterinary research, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
4 citing papers in PubMed.
- Article
- Avian pathogenic Escherichia coli virulence protein Hcp2a induces incomplete autophagy in chicken HD11 cells.Poultry science · 2026Article
- TheCells · 2026Article
- Pyrroloquinoline quinone protects heat-stressed porcine oocytes and improves early embryonic development via maintaining mitochondrial integrity and redox homeostasis.Biology direct · 2026Article
Corrections and comments
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Authors and funding
11 authors.
Funding
Abstract
Avian pathogenic Escherichia coli (APEC) is an extraintestinal pathogenic Escherichia coli that primarily causes avian colibacillosis, leading to localized or systemic infections. Its persistent survival within host macrophages is a critical component of the systemic infection process. In this process, outer membrane vesicles (OMVs) secreted by APEC play an important role; however, the interactions between OMVs and macrophages and their effects on bacterial intracellular survival remain unclear. This study demonstrated that APEC-derived OMVs induced intracellular Ca
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Registered trials
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