Evidence map›Paper›PMID 41583249›Full record

ArticleCureus2025

Once-Weekly Semaglutide Is Associated With Improvement in Vascular Endothelial Function in Patients With Type 2 Diabetes Mellitus: A Retrospective Observational Study.

Seigo Sugiyama, Akira Yoshida, Noboru Kurinami, Kunio Hieshima, Katsunori Jinnouchi, Tomoko Suzuki, Fumio Miyamoto, Keizo Kajiwara, Hideaki Jinnouchi

Abstract read
In one paragraph

Article in Cureus, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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0citing papers in PubMed
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1 · What the graph read from it

What it found

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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors.

Seigo SugiyamaDiabetes Care Center, Jinnouchi Hospital, Kumamoto, JPN.
Akira YoshidaDiabetes Care Center, Jinnouchi Hospital, Kumamoto, JPN.
Noboru KurinamiDiabetes Care Center, Jinnouchi Hospital, Kumamoto, JPN.
Kunio HieshimaDiabetes Care Center, Jinnouchi Hospital, Kumamoto, JPN.
Katsunori JinnouchiDiabetes Care Center, Jinnouchi Hospital, Kumamoto, JPN.
Tomoko SuzukiDiabetes Care Center, Jinnouchi Hospital, Kumamoto, JPN.
Fumio MiyamotoDiabetes Care Center, Jinnouchi Hospital, Kumamoto, JPN.
Keizo KajiwaraDiabetes Care Center, Jinnouchi Hospital, Kumamoto, JPN.
Hideaki JinnouchiDiabetes Care Center, Jinnouchi Hospital, Kumamoto, JPN.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundPatients with type 2 diabetes mellitus (T2DM) have a high-risk condition of developing cardiovascular disease, and therefore, therapeutic strategies should consider cardiovascular risk reduction. Previously, we demonstrated that circulating glucagon-like peptide-1 (GLP-1) levels are significantly lower in Japanese patients with coronary artery disease (CAD) compared with non-CAD subjects. Once-weekly semaglutide, a GLP-1 receptor agonist, has demonstrated clinical cardiometabolic benefits; however, its effect on vascular endothelial function in Japanese patients with T2DM remains unclear.

methodsThis retrospective observational study included patients with T2DM who were hospitalized at Jinnouchi Hospital and initiated once-weekly subcutaneous semaglutide therapy since 2021. Vascular endothelial function was assessed using reactive hyperemia peripheral arterial tonometry, expressed as the reactive hyperemia index (RHI), before and after semaglutide treatment. Clinical parameters including body weight (BW), glycated hemoglobin A1c (HbA1c), low-density lipoprotein cholesterol (LDL-cho), and high-sensitivity C-reactive protein (hsCRP) were also evaluated.

resultsA total of 24 patients (mean age: 55 years; n=20 (83.3%) male) were included. RHI significantly increased following semaglutide therapy (baseline: 1.62±0.19 versus follow-up: 2.04±0.60; p<0.01). BW, HbA1c, and LDL-cho also showed significant reductions (all p<0.01). HsCRP tended to decrease, though not significantly. None of the changes in BW, HbA1c, LDL-cho, or hsCRP correlated with the changes in RHI.

conclusionOnce-weekly semaglutide treatment significantly provided potential improvement in vascular endothelial function in Japanese patients with T2DM, supporting the cardiovascular protective role of semaglutide therapy in the clinical management of T2DM.

Indexed as

cardiovascular diseasesglucagon-like peptide-1 receptor agonistjapanese populationsemaglutidetype 2 diabetes mellitusvascular endothelial function

Identifiers

PMID41583249
PMCPMC12831498

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