ArticleNature metabolism2026
IL-21 mediates crosstalk between T cells and NK cells during the remission of type 1 diabetes.
Kang Lei, Xinyu Li, Ting Zhong, Rong Tang, Qiaolin Deng, Paul E Love, Zhiguang Zhou, Bin Zhao, Xia Li
Abstract read
In one paragraphArticle in Nature metabolism, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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What it found
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2 · The registryThe trial behind it
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5 · Who and what moneyAuthors and funding
9 authors.
Kang Lei *National Clinical Research Center for Endocrine and Metabolic Diseases, Key Laboratory of Diabetes Immunology, Ministry of Education, and Department of Metabolism and Endocrinology, The Second Xiangya Hospital of Central South University, Changsha, China.
Xinyu Li *National Clinical Research Center for Endocrine and Metabolic Diseases, Key Laboratory of Diabetes Immunology, Ministry of Education, and Department of Metabolism and Endocrinology, The Second Xiangya Hospital of Central South University, Changsha, China.
Ting Zhong *National Clinical Research Center for Endocrine and Metabolic Diseases, Key Laboratory of Diabetes Immunology, Ministry of Education, and Department of Metabolism and Endocrinology, The Second Xiangya Hospital of Central South University, Changsha, China.
Rong TangNational Clinical Research Center for Endocrine and Metabolic Diseases, Key Laboratory of Diabetes Immunology, Ministry of Education, and Department of Metabolism and Endocrinology, The Second Xiangya Hospital of Central South University, Changsha, China.
Paul E LoveSection on Hematopoiesis and Lymphocyte Biology, Eunice Kennedy Shriver National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, MD, USA.ORCID http://orcid.org/0000-0002-6682-7372 Zhiguang ZhouNational Clinical Research Center for Endocrine and Metabolic Diseases, Key Laboratory of Diabetes Immunology, Ministry of Education, and Department of Metabolism and Endocrinology, The Second Xiangya Hospital of Central South University, Changsha, China.
Bin ZhaoNational Clinical Research Center for Endocrine and Metabolic Diseases, Key Laboratory of Diabetes Immunology, Ministry of Education, and Department of Metabolism and Endocrinology, The Second Xiangya Hospital of Central South University, Changsha, China. binzhao@csu.edu.cn.ORCID http://orcid.org/0000-0003-4302-2333 Xia LiNational Clinical Research Center for Endocrine and Metabolic Diseases, Key Laboratory of Diabetes Immunology, Ministry of Education, and Department of Metabolism and Endocrinology, The Second Xiangya Hospital of Central South University, Changsha, China. lixia@csu.edu.cn.ORCID http://orcid.org/0000-0001-8665-7983 Funding
National Natural Science Foundation of China (National Science Foundation of China) 82170795National Natural Science Foundation of China (National Science Foundation of China) 82470814National Natural Science Foundation of China (National Science Foundation of China) 82470871National Science Foundation of China | National Natural Science Foundation of China-Yunnan Joint Fund (NSFC-Yunnan Joint Fund) 82501004
6 · The paper itselfAbstract
The innate immune system is increasingly recognized as a contributor to the development of type 1 diabetes (T1D), but the role of natural killer (NK) cells remains largely unclear. Here, we identify an expanded subset of transcriptionally active CD226
Indexed as
Diabetes Mellitus, Type 1InterleukinsKiller Cells, NaturalT-LymphocytesAnimalsFemaleHumansInterleukin-21MiceMice, Inbred NODSignal TransductionTOR Serine-Threonine KinasesInterleukin-21InterleukinsTOR Serine-Threonine Kinases
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