ArticleiScience2026
Ambient temperature exposure on inflammatory blood proteins-A longitudinal self-sampling survey in young Swedish adults.
Article in iScience, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper, 1 of them a synthesis that pooled it.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
1 citing paper in PubMed, 1 synthesis or guideline pooled it.
- Epigenetic Age Acceleration as a Modifiable Public Health Target: A Systematic Review and Meta-Analysis of Environmental, Behavioral, and Social Determinants with Development of the MEAB-Index.International journal of molecular sciences · 2026Pooled it
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
21 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
The mechanisms by which temperature exposure affects health outcomes remain unclear. In this study, we conducted three repeated measurements (2020-2022) by collecting self-sampled volumetric dry blood spots (DBSs) from 807 participants from the Swedish BAMSE cohort (mean age 25.9 years). By estimating individual-address level daily temperature using a high-resolution spatiotemporal model, we found that 58 (16%) of the 365 studied inflammation-related proteins were significantly associated with short-term exposure to ambient temperatures. The impact of temperature exposure was modified by sex, smoking, asthma, and concurrent exposure to air pollution. The temperature-associated proteins were linked to lung function, blood pressure, and HbA1c with validations in the UK Biobank. Furthermore, peak temperature exposure (both cold and heat) was associated with significantly increased proteomic age acceleration. Our findings suggest that ambient temperature exposure may cause adverse health effects through perturbating inflammation-related proteins.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.