Evidence map›Paper›PMID 41523060›Full record

ArticleJournal of pharmacy & bioallied sciences2025

An

Ruchi Agrawal, Mitesh Shah, Virag Srivastava, Shantala R Naik, Nikita R Patel, Shweta Dwivedy, Miral Mehta, Bhumika J Patel, Ramanpal Singh Makkad

Abstract read
In one paragraph

Article in Journal of pharmacy & bioallied sciences, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors.

Ruchi AgrawalDepartment of Microbiology, Bundelkhand Medical College, Sagar, Madhya Pradesh, India.
Mitesh ShahDepartment of Pathology, Bundelkhand Medical College, Sagar, Madhya Pradesh, India.
Virag SrivastavaDepartment of Dentistry, Government Medical College, Haldwani, Uttarakhand, India.
Shantala R NaikDepartment of Oral Medicine and Radiology, Dental Institute, Rajendra Institute of Medical Sciences, Ranchi, Jharkhand, India.
Nikita R PatelDepartment of Oral Pathology and Microbiology, SMBT Institute of Dental Sciences and Research, Nashik, Maharashtra, India.
Shweta DwivedyDepartment of Oral Medicine and Radiology, SMBT Institute of Dental Science and Research, Nashik, Maharashtra, India.
Miral MehtaDepartment of Pediatric and Preventive Dentistry, Karnavati School of Dentistry, Karnavati University, Gandhinagar, Gujarat, India.
Bhumika J PatelDepartment of Dentistry, Howard University, College of Dentistry, Washington, DC, USA.
Ramanpal Singh MakkadDepartment of Oral Medicine and Radiology New Horizon Dental College and Research Institute, Bilaspur, Chhattisgarh, India.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Background: OSCC stands as a prominent oral malignancy that develops because of lifestyle behavior and microbial activity. The pathogenesis of OSCC receives attention through modern research since Candida species reportedly contribute to the development of OSCC through inflammatory responses and nitrosamine generation and epithelial tissue malformation. Materials and Methods: Sixty subjects participated in this Results: Out of the 30 Group A samples, 80% (24/30) demonstrated candidal growth where Candida albicans constituted 70.8% of strains and Candida tropicalis made up 16.7% alongside Candida glabrata counting for 12.5%. The overall candidal growth among participants in Group B was limited to 30% (9/30) as C. albicans constituted 88.9%, whereas C. glabrata accounted for 11.1%. The incidence of candidal colonization proved markedly higher among the high-risk group compared to controls according to statistical analysis ( Conclusion: The numbers of Candida species, particularly C. albicans, rise substantially within the oral environments of patients dealing with potentially malignant disorders. The obtained data support the possibility that oral cancer development may be related to candidal colonization, thus demanding additional studies on this association.

Indexed as

Candida albicanscandidal colonizationCHROMagaroral microbiomeoral squamous cell carcinomapremalignant oral lesions

Identifiers

PMID41523060
PMCPMC12788484

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.