ArticleMolecular plant pathology2026
Transcription Factor PFB1 Is Required for the Botrytis cinerea Effector BcSCR1-Mediated Pathogenesis.
Article in Molecular plant pathology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
3 citing papers in PubMed.
- The Regulatory Army of Plant Defense: Transcription Factors in the War for Plant Immunity.International journal of molecular sciences · 2026Review
- The Moniliophthora roreri Effector MrB1 Affects Cellular Homeostasis and Induces Genotype-Dependent Necrosis in Theobroma cacao.Molecular plant pathology · 2026Article
- Transcription Factor PFB1 Is Required for the Botrytis cinerea Effector BcSCR1-Mediated Pathogenesis.Molecular plant pathology · 2026Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
9 authors.
Funding
Abstract
Botrytis cinerea is a necrotrophic fungus that infects more than 200 plant species, causing significant economic losses worldwide. Due to its diverse pathogenicity, extensive efforts have been made to unravel its pathogenic mechanisms. However, the function of the effectors underlying B. cinerea virulence remains largely unknown. In this study, we identified an effector BcSCR1 (B. cinerea small cysteine-rich protein 1) and found that it interacts with the transcription factor PFB1 (Pericycle Factor Type-B1) in Arabidopsis thaliana. Ectopic expression of BcSCR1 in plants resulted in decreased resistance to B. cinerea infection, while disruption of BcSCR1 in B. cinerea significantly decreased its aggressiveness toward host plants. In addition, the mutation of PFB1 in Arabidopsis increased resistance of plants to B. cinerea infection. PFB1 directly activated the expression of OPR3 and WRKY33 under normal conditions, while the binding of BcSCR1 to PFB1 repressed its transcriptional activation activity during B. cinerea infection. These findings reveal that B. cinerea employs the effector protein BcSCR1 to promote infection by suppressing the plant's immune defences.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.