ArticleMolecular psychiatry2026
Shared neurogenetic architecture links adolescent neurodevelopmental deviations to adult psychopathological procrastination.
Article in Molecular psychiatry, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
While general procrastination is common, psychopathological procrastination, a debilitating phenotype often indicative of subclinical psychiatric conditions, remains poorly understood in terms of its neurobiological underpinning. Challenging its traditional conceptualization as a mere behavioral deficit, we investigated the neurogenetic architecture of psychopathological procrastination. Leveraging a prospective adolescent twin cohort (N = 71 twin pairs) with neuroanatomical imaging (baseline) and psychopathological procrastination phenotyping in young adulthood (8-year follow-up), we first established moderate heritability for psychopathological procrastination (h² = 0.47, 95% CI: 0.14 - 0.71). Employing normative modeling of brain morphology, we found that adolescent neurodevelopmental deviations, specifically within the nucleus accumbens (NAcc), predicted adult psychopathological procrastination. Crucially, these predictive adolescent NAcc deviations exhibited a strong shared genetic basis with adult psychopathological procrastination (r
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