ArticleEcology letters2026
Drivers of Viral Prevalence in Landscape-Scale Pollinator Networks Across Europe: Honey Bee Viral Density, Niche Overlap With This Reservoir Host and Network Architecture.
Article in Ecology letters, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
3 citing papers in PubMed.
- Patterns of viral prevalence and load show independent circulation of some honey bee viruses in wild bees.Scientific reports · 2026Article
- Multiple Key Hosts and Network Structure Shape Viral Prevalence Across Multispecies Communities of Bees.Ecology letters · 2026Article
- Drivers of Viral Prevalence in Landscape-Scale Pollinator Networks Across Europe: Honey Bee Viral Density, Niche Overlap With This Reservoir Host and Network Architecture.Ecology letters · 2026Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
25 authors.
Funding
Abstract
Viral transfer from managed pollinators potentially threatens wild pollinators and may be exacerbated by land-use changes. Our causal models and plant-pollinator network data from 48 European urban and agricultural landscapes revealed the ecological mechanisms underpinning viral transmission. Host identity, network architecture and land-use modulated viral dynamics (black queen cell virus, BQCV; deformed wing virus, DWV-A and DWV-B). Viral prevalence in wild pollinators was driven by viral density in the reservoir host: honey bees, and secondarily by trophic niche overlap with these managed pollinators. Modular networks limited BQCV prevalence, which was driven by reduced honey bee niche overlap, suggesting minimal onward transmission among wild pollinators. Landscapes supporting greater wild pollinator abundance diluted DWV-B transmission; in urban landscapes managed honey bees and wild pollinators experienced higher and lower BQCV prevalence, respectively. Disease in managed bee colonies and land-use changes that concentrate pollinator foraging interactions present potential viral risks to wild pollinator health.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.