ArticleCureus2025
Subendocardial Late Gadolinium Enhancement Without Troponin Elevation: A Case of Apical Hypertrophic Cardiomyopathy With Myocardial Involvement.
Article in Cureus, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Authors and funding
4 authors.
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Abstract
Apical hypertrophic cardiomyopathy (AHCM), also known as Yamaguchi syndrome, is a rare variant of hypertrophic cardiomyopathy (HCM) characterized by localized thickening of the left ventricular apex. Clinically, it may mimic acute coronary syndrome (ACS), myocarditis, or pericarditis. Cardiac magnetic resonance (CMR) allows the identification of myocardial involvement even in the absence of elevated serum biomarkers. A 39-year-old Hispanic man presented with a four-day history of oppressive chest pain radiating to the left arm, associated with dyspnea, fever, and dysuria. On admission, ECG demonstrated deep T-wave inversions; initial risk stratification (a thrombolysis in myocardial infarction (TIMI) score of 3 and a history, ECG, age, risk factors, and troponin (HEART) score of 4) indicated intermediate probability of ACS. hs-cTn remained negative; however, laboratory tests showed leukocytosis with neutrophilia, elevated inflammatory markers, and a positive urine culture for
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