ArticleScientific reports2025
Auditory genotype-phenotype correlation of patients with variants in STRC.
Article in Scientific reports, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
1 citing paper in PubMed.
- Expression of key genes in IVIG-unresponsive Kawasaki disease and their immune correlation based on bioinformatics analysis.Immunologic research · 2026Article
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Authors and funding
7 authors.
Funding
Abstract
Pathogenic variants in the STRC gene are among the most common causes of autosomal recessive non-syndromic hearing loss, particularly in cases with mild-to-moderate sensorineural hearing loss (SNHL). Despite its prevalence, the clinical phenotype and natural history of STRC-related SNHL remain undercharacterized due to diagnostic challenges posed by a highly homologous pseudogene, pSTRC. This study included 23 families enrolled in the Yonsei University Hearing Loss cohort. Genetic testing was performed using either targeted deafness gene panels or whole-exome sequencing, followed by multiplex ligation-dependent probe amplification and confirmatory Sanger sequencing. A total of 23 patients with STRC-related SNHL were identified, including 12 with homozygous STRC/CATSPER2 gene deletions and 11 with other combinations of pathogenic variants. Most patients exhibited mild-to-moderate SNHL with flat or gently sloping audiometric configurations, predominantly affecting mid-to-high frequencies. No significant differences in mean PTA thresholds were observed between the two genotypic groups. Longitudinal analysis over a follow-up period of up to 4 years demonstrated stable hearing thresholds in 75% of ears, with no significant progression detected using linear mixed model analysis. Linear regression showed no age-dependent threshold shift in either ear across all genotypic subgroups. In conclusion, STRC-related hearing loss is typically mild-to-moderate, stable over time, and audiometrically similar regardless of genotypic subclassification. Given its subtle phenotype and diagnostic complexity, STRC mutations may be underrecognized without targeted screening. Incorporating STRC-specific MLPA assay into routine genetic diagnostics in patients with mild-to-moderate hearing loss may improve early detection and guide timely precision intervention.
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