ArticlePlant communications2026
The CCCH-tandem zinc-finger protein OsTZF1 positively regulates basal immunity but suppresses Piz-t-mediated blast resistance in rice.
Article in Plant communications, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. An erratum has been issued. Cited by 1 paper.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
1 citing paper in PubMed.
- Jacalin-Related LectinPlants (Basel, Switzerland) · 2026Article
Corrections and comments
- Erratum issued
Authors and funding
13 authors.
Funding
Abstract
Plant nucleotide-binding leucine-rich repeat (NLR) immune receptors play a critical role in plant defense, but the mechanisms regulating their expression remain incompletely understood. Here, we report that the rice CCCH-tandem zinc-finger protein OsTZF1 interacts with both AvrPiz-t interacting protein 10 (APIP10) and the NLR protein Piz-t. OsTZF1 positively regulates rice immunity by increasing the transcript levels of OsSGT1 and OsKS4 through binding to the AAAGC motif. However, in Piz-t-carrying plants, knockout of OsTZF1 results in increased Piz-t transcript and protein levels, thus enhancing resistance to virulent strains of Magnaporthe oryzae without a growth penalty. Furthermore, we demonstrate that OsTZF1 can directly bind to the AAAGC motif of Piz-t, thereby repressing its expression level. Thus, OsTZF1 functions as a dual regulator: it activates general defense genes while suppressing Piz-t-mediated resistance to virulent strains, balancing resistance and growth. These findings reveal a strategy for engineering NLR-mediated resistance in crops by fine-tuning immune regulators to achieve both disease resistance and yield stability.
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