ReviewMolecular plant pathology2025
Current Status and Future Direction of the High-Temperature Resistance in Wheat to Control Stripe Rust.
Review in Molecular plant pathology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
2 citing papers in PubMed.
- The Wheat CRK-RLCK-MAPKs Signalling Module Confers High-Temperature All-Stage Resistance to Stripe Rust.Plant biotechnology journal · 2026Article
- Current Status and Future Direction of the High-Temperature Resistance in Wheat to Control Stripe Rust.Molecular plant pathology · 2025Review
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
10 authors.
Funding
Abstract
This review summarises the current advances and future perspectives on high-temperature resistance in wheat against stripe rust, caused by the airborne fungal pathogen Puccinia striiformis f. sp. tritici (Pst). High-temperature resistance, comprising high-temperature adult-plant (HTAP) and high-temperature all-stage (HTAS) resistance, is critical for durable disease control. HTAP resistance occurs in adult plants at relatively high temperatures, whereas HTAS resistance acts across all growth stages at relatively high temperatures. Genetic mapping has identified numerous HTAP-resistance genes and quantitative trait loci (QTLs) in various chromosomal regions, especially in the B subgenome. Cloned genes, such as Yr18, Yr36 and Yr46, reveal mechanisms involving cell necrosis induction, photosynthesis modulation and nutrient transportation. For HTAS resistance, defence-related factors such as nucleotide-binding site-leucine-rich repeat proteins, transcription factors and receptor-like kinases that mediate resistance via plant-pathogen interactions have been identified; however, research on identifying the genes or QTLs that can be used in breeding programmes is limited. To overcome host defence, Pst secretes effectors that suppress high-temperature resistance by targeting host proteins. Future research should focus on novel gene mining, regulatory network deciphering, effector-host interaction studies and breeding applications via marker-assisted selection and gene editing.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.