Evidence map›Paper›PMID 41404359›Full record

ArticleMedical research archives2025

Neuroinflammatory Mechanisms and Vocalization Biomarkers in the

Sarah A Lechner, Cynthia A Kelm-Nelson

Abstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Medical research archives, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Sarah A LechnerDepartment of Surgery, Division of Endocrine Surgery, University of Wisconsin, Madison, Wisconsin, USA.
Cynthia A Kelm-NelsonDepartment of Surgery, Division of Endocrine Surgery, University of Wisconsin, Madison, Wisconsin, USA.

Funding

Pathways, mechanisms, and treatments of vocal communication deficits in a Parkinson rat modelR01NS117469 · NINDS · UNIVERSITY OF WISCONSIN-MADISON · PI Cynthia A Kelm-Nelson · 2020 to 2026
$3.0M
NINDS NIH HHS R01 NS117469
6 · The paper itself

Abstract

Parkinson's disease is a progressive neurodegenerative disorder with an unclear etiology and a long preclinical phase. The multitude of challenges of studying this in humans highlight the need for robust preclinical animal models that accurately reflect the disease's preclinical progression. While traditional neurotoxin models fail to capture the slow, genetic nature of PD, genetic models, such as the phosphatase and tensin homolog-induced putative kinase 1 (

Indexed as

biomarkerneuroinflammationParkinson diseaseratultrasonic vocalization

Identifiers

PMID41404359
PMCPMC12703701

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.