ArticleAnnals of medicine and surgery (2012)2025
From vision loss to paralysis: a striking case of Wernicke's encephalopathy in pregnancy.
Article in Annals of medicine and surgery (2012), 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Abstract
Introduction: Wernicke's encephalopathy (WE) is a neurological emergency caused by thiamine deficiency, most often associated with chronic alcoholism, but it can also occur in non-alcoholic settings such as hyperemesis gravidarum. Case presentation: We report the case of a 37-year-old pregnant woman with intractable vomiting who developed blurred vision, ophthalmoplegia, and subsequently acute flaccid paraparesis. Brain MRI showed typical lesions of WE, and laboratory tests revealed profound hypokalemia. Clinical improvement followed intravenous thiamine and potassium supplementation. Clinical discussion: This case illustrates the dual neurological complications of hyperemesis gravidarum: Wernicke's encephalopathy and hypokalemic paralysis, both resulting from severe nutritional and electrolyte depletion. Conclusion: Hyperemesis gravidarum can cause both thiamine deficiency and severe hypokalemia. Early recognition and combined correction are essential to prevent permanent neurological sequelae.
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