ArticleScientific reports2025
Development of urinary impairment after spinal cord injury courses with altered urethral serotonin signalling in the female mice.
Article in Scientific reports, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
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Who cites it
2 citing papers in PubMed.
- Effect of intrasphincteric botulinum toxin on postoperative urinary retention following stapled hemorrhoidopexy: a randomized, double-blind, placebo-controlled trial.Scientific reports · 2026Trial
- Engineering functional vasculatures to reconstruct sympathetic-parasympathetic circuits for bladder function after spinal cord injury.Bioactive materials · 2027Article
Corrections and comments
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Authors and funding
5 authors.
Funding
Abstract
Spinal Cord Injury (SCI) often leads to urinary incontinence. While alterations in the bladder have been considered the primary cause of post-SCI urinary dysfunction, evidence suggests that urethral afferents may influence post-SCI bladder activity. These afferents respond to locally produced serotonin (5-HT), and 5-HT signalling may likely be affected by SCI. Here, we investigated the role of urethral 5-HT on post-SCI bladder reflex activity using female C57BL/6 mice (wild-type -WT- and animals lacking urethral 5-HT (tryptophan hydroxylase 1 -tph1- null -tph1
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