Evidence map›Paper›PMID 41351260›Full record

ArticlePhysiological reports2025

Strategies for prolonging ventricular action potential duration without increasing transmural dispersion of repolarization.

Candido Cabo

Abstract read
In one paragraph

Article in Physiological reports, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Article
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author.

Candido CaboDepartment of Computer Systems, New York City College of Technology, New York, New York, USA.ORCID 0000-0002-5119-981X

Funding

Professional Staff Congress (PSC) of the City University of New York (CUNY) 67024-0055
6 · The paper itself

Abstract

Dispersion of repolarization results from a non-homogeneous recovery of excitability in cardiac tissue, and it is an important factor in arrhythmogenesis because it could lead to the initiation and maintenance of a variety of arrhythmias. Antiarrhythmic agents that prolong action potential duration (APD) by selectively blocking specific ion channels (like I

Indexed as

Action PotentialsAnti-Arrhythmia AgentsModels, CardiovascularMyocytes, CardiacArrhythmias, CardiacComputer SimulationHeart VentriclesHumansIon ChannelsAnti-Arrhythmia AgentsIon Channelscomputer modelsdispersion of repolarizationion channelsmultichannel pharmacology

Identifiers

PMID41351260
PMCPMC12680787

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.