Evidence map›Paper›PMID 41322438›Full record

ArticleOpen life sciences2025

Genetic variants in VWF exon 26 and their implications for type 1 Von Willebrand disease in a Saudi Arabian population.

Faisal M Alzahrani, Asma A Al Faris, Layla A Bashawri, Maryam A Aldossary, Osama Al Sultan, Ahmed Mousa Alzahrani, Thekra N Al-Maqati, Elmoeiz A Elnagi, Fathelrahman Mahdi Hassan

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Article in Open life sciences, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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1 · What the graph read from it

What it found

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2 · The registry

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3 · Its place in the literature

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4 · The record

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5 · Who and what money

Authors and funding

9 authors.

Faisal M AlzahraniDepartment of Clinical Laboratory Sciences, College of Applied Medical Sciences, Imam Abdulrahman Bin Faisal University, Dammam, Saudi Arabia.ORCID https://orcid.org/0000-0003-4425-6502
Asma A Al FarisDepartment of Clinical Laboratory Sciences, College of Applied Medical Sciences, Imam Abdulrahman Bin Faisal University, Dammam, Saudi Arabia.ORCID https://orcid.org/0000-0003-2473-7421
Layla A BashawriDepartment of Clinical Laboratory Sciences, College of Applied Medical Sciences, Imam Abdulrahman Bin Faisal University, Dammam, Saudi Arabia.ORCID https://orcid.org/0000-0003-0557-4954
Maryam A AldossaryDepartment of Clinical Laboratory Sciences, College of Applied Medical Sciences, Imam Abdulrahman Bin Faisal University, Dammam, Saudi Arabia.ORCID https://orcid.org/0000-0002-0435-4709
Osama Al SultanDepartment of Internal Medicine, King Fahad Hospital of the University, Imam Abdulrahman Bin Faisal University, Khobar, Saudi Arabia.ORCID https://orcid.org/0000-0002-4556-2213
Ahmed Mousa AlzahraniBlood Bank, Department of Pathology and Laboratory Medicine, King Abdulaziz Medical City in Jeddah, Ministry of National Guard, Jeddah, Saudi Arabia.ORCID https://orcid.org/0009-0002-3986-6330
Thekra N Al-MaqatiDepartment of Clinical Laboratory Sciences, Prince Sultan Military College of Health Sciences, Dhahran, Saudi Arabia.ORCID https://orcid.org/0000-0001-5705-2053
Elmoeiz A ElnagiDepartment of Clinical Laboratory Sciences, Prince Sultan Military College of Health Sciences, Dhahran, Saudi Arabia.ORCID https://orcid.org/0000-0003-1352-1350
Fathelrahman Mahdi HassanDepartment of Hematology and Immunohematology, College of Medical Laboratory Science, Sudan University of Science and Technology, Khartoum, Sudan.ORCID https://orcid.org/0000-0002-2630-3208

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Von Willebrand disease (VWD) is one of the most common bleeding disorders, stemming from irregularities in the Von Willebrand factor (VWF). Globally, type 1 VWD (VWD1) is the most prevalent form, characterized by decreased levels of VWF in the blood. Genotyping studies have found causative genetic variants in approximately 65% of VWD1 cases, revealing the presence of ethnic-specific VWF variants. This study is limited by its exclusive focus on exon 26, warranting further investigation of other exons and the inclusion of functional analyses. Moreover, these findings emphasize the importance of understanding genetic variability in population-specific contexts, supporting the necessity for future studies on additional exons and potential epigenetic interactions. Between 2018 and 2019, samples and relevant medical data were collected at King Fahad Hospital of the University in Al Khobar. Sanger sequencing of the exonic and flanking intronic regions of exon 26 was performed on 22 index cases clinically diagnosed with VWD1 and their first-degree relatives. Analysis revealed four exonic and 11 intronic variants in VWF exon 26 among the participants. None of the identified variants appeared to explain the VWD-related clinical features in these individuals. However, findings suggest a higher prevalence of specific VWF variants within the Saudi population compared to global databases. Further investigation, including sequencing of additional exons or next-generation sequencing, may help to uncover potential disease-causing variants in this cohort. Establishing a national sequencing project could also enhance our understanding of both common and rare variants in the genetic basis of complex diseases, such as VWD1.

Indexed as

exon 26sanger sequencingVon Willebrand Disease type 1Von Willebrand factor

Identifiers

PMID41322438
PMCPMC12658733

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.