ArticleBMC plant biology2025
Analysis of physiological and molecular mechanisms of cotton trichome aphid resistance based on SWS-1 mutant.
Article in BMC plant biology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed.
No citing paper in PubMed yet.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
10 authors.
Funding
Abstract
Cotton is a globally significant cash crop; however, its production is frequently compromised by aphid infestations. This study systematically investigated the regulatory mechanisms governing aphid resistance through leaf trichome development. A comprehensive investigation, encompassing multi-omics analysis, genetic validation, and physiological-biochemical experiments, was conducted to determine the impact of trichome density on aphid resistance. The findings revealed that trichome density exceeding 701.4/cm² significantly enhances aphid resistance. This mechanism involves the creation of physical barriers to aphid feeding and the induction of defensive metabolite accumulation (e.g., β-caryophyllene and D-limonene). After aphid feeding, there was a significant increase in soluble sugar content and POD activity in the high-trichome lines. Subsequent analysis, employing both BSA-seq and transcriptomic techniques, has identified four key genes (GhABCG32, GhV6Z11, GhRSD1, and GhSHMT3). The functional validation experiment demonstrated that the silencing of the GhABCG32 gene via the VIGS (virus-induced gene silencing) technology resulted in a significant reduction in leaf trichomes and a substantial decrease in stem trichomes. This finding serves to confirm the critical role of this gene in trichome formation and aphid resistance. The present study provides theoretical foundations and key gene targets for molecular design breeding of high-yielding aphid-resistant cotton varieties.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.