ArticleCureus2025
Amiodarone-Induced Pulmonary Toxicity With Severe Pulmonary Fibrosis Complicated by Stenotrophomonas maltophilia Pneumonia: A Fatal Case Report and Literature Review.
Article in Cureus, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
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0 citing papers in PubMed.
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Authors and funding
6 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Amiodarone is a widely used antiarrhythmic agent, valued for its efficacy and low proarrhythmic risk, but it carries a known potential for serious toxicities, particularly pulmonary toxicity. Amiodarone-induced pulmonary toxicity (APT) remains a life-threatening adverse effect. We report the case of a 75-year-old male with a 30-year history of amiodarone use (200 mg twice daily) who developed progressive respiratory distress consistent with APT. Despite treatment with broad-spectrum antibiotics and corticosteroids, his condition deteriorated. Imaging and clinical findings raised suspicion for APT, which was managed with amiodarone discontinuation and pulse-dose steroid therapy. Subsequently, he developed hospital-acquired
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