Evidence map›Paper›PMID 41269421›Full record

ReviewMolecular neurobiology2025

Targeting Neurodegeneration with SGLT2is: From Molecular Mechanisms to Clinical Implications.

Hamideh Asadinejad, Soroush Taherkhani, Sakine Mosaei Golboos, Yaser Azizi, Ali Mohammadkhanizadeh

Abstract readReview
PubMed Publisher
In one paragraph

Review in Molecular neurobiology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed.

  1. Review
  2. Article
  3. Review
  4. Review
  5. Review
  6. Article
  7. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Hamideh AsadinejadDepartment of Physiology, School of Medicine, Iran University of Medical Sciences, Tehran, Iran.
Soroush TaherkhaniDepartment of Physiology, School of Medicine, Iran University of Medical Sciences, Tehran, Iran.
Sakine Mosaei GolboosDepartment of Physiology, School of Medicine, Iran University of Medical Sciences, Tehran, Iran.
Yaser AziziDepartment of Physiology, School of Medicine, Iran University of Medical Sciences, Tehran, Iran. azizi.y@iums.ac.ir.
Ali MohammadkhanizadehDepartment of Physiology, School of Medicine, Iran University of Medical Sciences, Tehran, Iran. Ali_mohamadkhany@yahoo.com.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Sodium-glucose cotransporter-2 inhibitors (SGLT2is) are a class of antidiabetic drugs that have demonstrated significant cardiovascular and renal benefits. Accumulating evidence suggests that SGLT2is also exert neuroprotective effects and may influence the progression of neurodegenerative diseases such as Alzheimer's disease (AD), Parkinson's disease (PD), and amyotrophic lateral sclerosis (ALS). SGLT2is modulate glucose metabolism, reduce oxidative stress, suppress inflammation, and enhance mitochondrial function. Beyond glycemic control, they show potential therapeutic effects in ameliorating the metabolic dysregulation associated with neurodegenerative pathologies. Current preclinical and clinical evidence including metabolic regulation, anti-inflammatory actions, and neuroprotective effects mediated through SGLT2is associated molecular pathways have been critically evaluated to delineate their therapeutic potential in neurodegenerative disorders. Although preclinical studies have shown promising results, more clinical trials are needed. This review highlights key research gaps and proposes future translational applications.

Indexed as

Neurodegenerative DiseasesNeuroprotective AgentsSodium-Glucose Transporter 2 InhibitorsAnimalsHumansNeuroprotective AgentsSodium-Glucose Transporter 2 InhibitorsInflammationMetabolicMitochondriaNeuroprotectionSGLT2 inhibitor

Identifiers

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.