Evidence map›Paper›PMID 41268609›Full record

ArticleMolecular medicine reports2026

High‑intensity exercise training inhibits excessive autophagy in the hyperlipidemic myocardium of ApoE

Shan Gao, Wei Yao, Jin Yang, Yujie Liu, Zuowei Pei

Abstract read
In one paragraph

Article in Molecular medicine reports, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Shan Gao *Department of Central Laboratory, Central Hospital of Dalian University of Technology, Dalian, Liaoning 116033, P.R. China.
Wei Yao *Department of Ward of Emergency Internal Medicine, Central Hospital of Dalian University of Technology, Dalian, Liaoning 116033, P.R. China.
Jin YangDepartment of Central Laboratory, Central Hospital of Dalian University of Technology, Dalian, Liaoning 116033, P.R. China.
Yujie LiuDepartment of Central Laboratory, Central Hospital of Dalian University of Technology, Dalian, Liaoning 116033, P.R. China.
Zuowei PeiDepartment of Central Laboratory, Central Hospital of Dalian University of Technology, Dalian, Liaoning 116033, P.R. China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Hyperlipidemia represents a key contributory factor in the development and progression of cardiovascular diseases, contributing to cardiac injury through mechanisms involving oxidative stress, inflammation and autophagic cell death. Nicotinamide adenine dinucleotide (NAD

Indexed as

Apolipoproteins EAutophagyHyperlipidemiasMyocardiumNADPhysical Conditioning, AnimalSirtuin 1AnimalsDiet, High-FatDisease Models, AnimalMaleMiceMice, Inbred C57BLOxidative StressSignal TransductionApolipoproteins ENADSirt1 protein, mouseSirtuin 1apolipoprotein E‑deficient miceautophagycardiac damagehyperlipidemianicotinamide adenine dinucleotide

Identifiers

PMID41268609
PMCPMC12676213

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.