Evidence map›Paper›PMID 41264038›Full record

ReviewMolecular neurobiology2025

The Potential Roles of HSPA2 in Parkinson's Disease.

Cheng Li, Shu Li, Yi Lu, Yu Zhu, Renshi Xu

Abstract readReview
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In one paragraph

Review in Molecular neurobiology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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0citing papers in PubMed
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1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

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3 · Its place in the literature

Who cites it

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No citing paper in PubMed yet.

4 · The record

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5 · Who and what money

Authors and funding

5 authors.

Cheng Li *Department of Neurology, Jiangxi Provincial People's Hospital, The First Affiliated Hospital of Nanchang Medical College, The Clinical College of Nanchang Medical College, National Regional Center for Neurological Diseases, Xiangya Hospital of Central South University Jiangxi Hospital, Nanchang, 330006, Jiangxi, China.
Shu Li *Department of Neurology, Jiangxi Provincial People's Hospital, The First Affiliated Hospital of Nanchang Medical College, The Clinical College of Nanchang Medical College, National Regional Center for Neurological Diseases, Xiangya Hospital of Central South University Jiangxi Hospital, Nanchang, 330006, Jiangxi, China.
Yi Lu *Department of Health Management Center, The First Affiliated Hospital, Jiangxi Medical College, Nanchang University, Nanchang, 330006, Jiangxi, China.
Yu ZhuDepartment of Neurology, The First Affiliated Hospital, Jiangxi Medical College, Nanchang University, Nanchang, 330006, Jiangxi, China. zhuyu19930703@163.com.
Renshi XuDepartment of Neurology, Jiangxi Provincial People's Hospital, The First Affiliated Hospital of Nanchang Medical College, The Clinical College of Nanchang Medical College, National Regional Center for Neurological Diseases, Xiangya Hospital of Central South University Jiangxi Hospital, Nanchang, 330006, Jiangxi, China. xurenshi@ncu.edu.cn.

Funding

Education Department of Jiangxi Province GJJ13198, GJJ170021, 2200115Jiangxi Province Key Laboratory of Neurology 2024SSY06081Jiangxi Provincial Department of Science and Technology 20142BBG70062, 20171BAB215022, 20192BAB205043Jiangxi Provincial Health and Health Commission 202210002, 202310119National Natural Science Foundation of China 30560042, 81160161, 81360198, 82160255
6 · The paper itself

Abstract

Parkinson's disease (PD) is a neurodegenerative disorder traditionally identified by its characteristic motor symptoms, the presence of Lewy bodies, and the loss of neurons in the substantia nigra. At present, there is no cure for the disease. Our study aimed to uncover the new aspects of the pathogenesis underlying PD. Firstly, we identified that HSPA2 is potentially involved in the PD pathogenesis through a bioinformatic approach; HSPA2 was associated with the disease duration. For that, we analyzed the correlation of the clinical information of patients with the HSPA2 gene expression level. Then, the expression of HSPA2 was investigated in vivo using the midbrain from the MPTP-treated mouse models. The HSPA2 levels significantly increased in the midbrain tissue from the PD mouse models. The mechanistic studies about HSPA2 in the pathogenesis of PD were conducted in vitro in the HEK293T cell line, which observed the changes in α-synuclein expression by regulating the HSPA2 expression. In the HEK293T cell line, the overexpression of HSPA2 increased the α-synuclein aggregates formation, and the knockdown of HSPA2 reduced the α-synuclein aggregates accumulation. Therefore, this study hypothesized that HSPA2 might be a new regulator of α-synuclein associated with the PD pathology.

Indexed as

HSP70 Heat-Shock ProteinsParkinson Diseasealpha-SynucleinAnimalsDisease Models, AnimalFemaleHEK293 CellsHumansMaleMesencephalonMiceMice, Inbred C57BLProtein Aggregatesalpha-SynucleinHSP70 Heat-Shock ProteinsHSPA2 protein, humanProtein AggregatesBioinformatic analysisHEK293T cellHSPA2MPTP-treated mouse modelParkinson’s diseaseα-synuclein

Identifiers

PMID41264038

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.