ArticleJACC. Case reports2026
Recurrent Ventricular Arrhythmia From Acquired Long QT Syndrome Due to Alcohol-Related Electrolyte Depletion.
Article in JACC. Case reports, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
backgroundElectrolyte depletion during alcohol withdrawal can cause acquired long QT syndrome (LQTS), leading to torsades de pointes (TdP) and ventricular fibrillation (VF) CASE SUMMARY: A 64-year-old man with a history of heavy alcohol use presented with syncope. Evaluation revealed a prolonged QTc interval of 511 ms, along with hypokalemia (K DISCUSSION: Alcohol withdrawal can cause marked QTc prolongation via electrolyte disturbances, precipitating TdP and VF. This under-recognized complication highlights the need for vigilant monitoring and prompt electrolyte correction to prevent fatal arrhythmias. TAKE-HOME MESSAGES: In acute alcohol withdrawal syndrome , severe hypokalemia and hypomagnesemia can induce acquired LQTS, resulting in TdP and VF. Management includes defibrillation, magnesium and potassium repletion, and avoiding QT-prolonging drugs. Implantable cardioverter-defibrillator placement is not indicated once electrolyte abnormalities are corrected.
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