ArticleScience advances2025
Transition of pseudorabies virus from latency to reactivation state selectively triggered by pathogenic bacteria.
Article in Science advances, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
3 citing papers in PubMed.
- Isoliquiritigenin Elicits Potent Antiviral Activity Against Pseudorabies Virus Through Modulating the cGAS/STING and JAK/STAT Signaling Cascades.Veterinary sciences · 2026Article
- Article
- A Humanized Anti-gD Broadly Neutralizing Antibody Confers Complete Post-Exposure Protection Against Pseudorabies Virus.Advanced science (Weinheim, Baden-Wurttemberg, Germany) · 2026Article
Corrections and comments
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Authors and funding
12 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Pseudorabies virus (PRV) causes severe morbidity and mortality in pigs and must be eradicated from pig populations. The increasing PRV infections in humans-resulting in severe encephalitis, neurological sequelae, and death since 2017 in China-have also posed a public health risk. PRV can establish latency in pigs, potentially leading to disease outbreaks upon reactivation. However, little is known about the PRV reactivation process, particularly in cases with other pathogens coinfection. We found that extraintestinal pathogenic
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