ArticleNaunyn-Schmiedeberg's archives of pharmacology2026
Coenzyme Q10 protects against glyphosate-based herbicide-induced testicular and sperm toxicity by modulating Nrf2/Keap1/HO-1 and TLR4/NF-κB pathways.
Article in Naunyn-Schmiedeberg's archives of pharmacology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
3 citing papers in PubMed.
- Review
- Modulation of P2X7-driven purinergic signaling and NLRP3 inflammasome activation underlies the hepatoprotective effects of coenzyme Q10 against glyphosate-based herbicide exposure.Purinergic signalling · 2026Article
- Protective effects of multi-strain probiotics against polystyrene nanoplastic-induced parotid gland toxicity: role of NRF2/HO-1 and NF-κB pathways.Frontiers in pharmacology · 2026Article
Corrections and comments
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Authors and funding
7 authors.
Funding
Abstract
Roundup, the most widely used glyphosate-based herbicide (GBH), contains glyphosate (GLY) as its main active ingredient and was used to model GBH-induced reproductive toxicity in this study. We aimed to investigate the protective effect of coenzyme Q10 (CoQ10) against GLY-induced testicular and sperm damage, focusing on oxidative stress, inflammation, and apoptosis. Unlike previous studies, this work provides the first integrated evidence combining histopathological, biochemical, hormonal, and molecular assessments with functional sperm analyses, and uniquely demonstrates how CoQ10 modulates both antioxidant (Nrf2/Keap1/HO-1) and inflammatory (TLR4/NF-κB) pathways in GBH-induced testicular injury. Forty-two adult male ICR mice were assigned to five groups: control, sham, CoQ10 (200 mg/kg/day), GLY (500 mg/kg/day), and GLY + CoQ10 (500 + 200 mg/kg/day). The treatments were administered orally for 35 days, corresponding to one spermatogenesis cycle. GLY reduced testicular weight, disrupted histoarchitecture, increased oxidative stress (↑MDA, TOS, OSI; ↓GSH, TAS), suppressed Nrf2/Keap1/HO-1 signaling, upregulated TLR4/NF-κB, and triggered apoptosis (↑Bax, ↓Bcl-2). It also impaired sperm parameters (concentration, motility, morphology, viability, membrane integrity) and increased mitochondrial ROS, while reducing testosterone and elevating LH. CoQ10 co-treatment alleviated these alterations by restoring redox balance, normalizing signaling pathways, improving sperm quality, and correcting hormonal changes. In conclusion, CoQ10 mitigates GLY-induced testicular toxicity by modulating oxidative stress, inflammation, and apoptosis, highlighting its novel therapeutic potential for protecting male fertility against GBH exposure.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.