ArticleNature communications2025
A fungal pathogen manipulates phytocytokine signaling for plant infection.
Article in Nature communications, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
5 citing papers in PubMed.
- Veh1 modulates CaaBIOTECH · 2026Article
- Multiple BnaSOBIR1-Associated Receptor-like Proteins Contribute to Enhanced Resistance toPlants (Basel, Switzerland) · 2026Article
- PEPR1 Mediates SsNLP1-Triggered Immunity AgainstInternational journal of molecular sciences · 2026Article
- Processing and release of endogenous immunogenic peptide signals.The Plant journal : for cell and molecular biology · 2026Review
- Plant-parasitic nematodes produce functional mimics of plant PSK peptides to facilitate parasitism.bioRxiv : the preprint server for biology · 2026Article
Corrections and comments
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Authors and funding
6 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Phytocytokines, hormone-like plant peptides, play crucial roles in immune regulation and development. Phytosulfokine (PSK), known to mediate plant growth, also modulates plant pattern-triggered immunity (PTI). Here, we demonstrate that VdSCP8, a small cysteine-containing effector from Verticillium dahliae, functions as a virulence-promoting protein that suppresses PTI in Arabidopsis thaliana and Nicotiana benthamiana. Apoplastic SCP8 suppresses immune activation mediated by leucine-rich repeat ectodomain pattern recognition receptors. SCP8 virulence and immunosuppressive activities require PHYTOSULFOKINE RECEPTOR 1 (PSKR1), which binds PSK and forms a complex with co-receptor BAK1 to suppress PTI. Our findings underscore SCP8's role in PTI suppression, facilitating PSKR1-BAK1 complex formation and promoting PSK accumulation through a plant subtilase. These results highlight how a multi-host plant pathogen manipulates PTI through enhancing immunosuppressive PSK signaling.
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Registered trials
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