Evidence map›Paper›PMID 41226391›Full record

ArticleInternational journal of molecular sciences2025

Anti-Oxidative and Anti-Inflammatory Effect of Protaetia Brevitarsis-Derived Protein Hydrolysates in Adipose Tissues of Obese Mice.

Jun-Koo Kang, Eun Hye Lee, Bo Hyun Yoon, Minji Jeon, Jae-Wook Chung, Phil Hyun Song, Tae Gyun Kwon, Yun-Sok Ha, So Young Chun, Syng-Ook Lee and 1 more

Abstract read
In one paragraph

Article in International journal of molecular sciences, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors.

Jun-Koo KangDepartment of Urology, School of Medicine, Kyungpook National University, Daegu 41566, Republic of Korea.
Eun Hye LeeJoint Institute for Regenerative Medicine, Kyungpook National University, Daegu 41566, Republic of Korea.
Bo Hyun YoonJoint Institute for Regenerative Medicine, Kyungpook National University, Daegu 41566, Republic of Korea.
Minji JeonJoint Institute for Regenerative Medicine, Kyungpook National University, Daegu 41566, Republic of Korea.
Jae-Wook ChungDepartment of Urology, School of Medicine, Kyungpook National University, Daegu 41566, Republic of Korea.ORCID 0000-0002-1055-2357
Phil Hyun SongDepartment of Urology, College of Medicine, Yeungnam University, Daegu 42415, Republic of Korea.
Tae Gyun KwonDepartment of Urology, School of Medicine, Kyungpook National University, Daegu 41566, Republic of Korea.
Yun-Sok HaDepartment of Urology, School of Medicine, Kyungpook National University, Daegu 41566, Republic of Korea.ORCID 0000-0003-3732-9814
So Young ChunJoint Institute for Regenerative Medicine, Kyungpook National University, Daegu 41566, Republic of Korea.
Syng-Ook LeeDepartment of Food Science and Technology, Keimyung University, Daegu 42403, Republic of Korea.ORCID 0000-0002-8958-8634
Bum Soo KimDepartment of Urology, School of Medicine, Kyungpook National University, Daegu 41566, Republic of Korea.ORCID 0000-0002-4873-3049

Funding

This work was supported by Biomedical Research Institute grant, Kyungpook National University Hospital 2022
6 · The paper itself

Abstract

Obesity is a major global health issue linked to metabolic disorders, chronic inflammation, and systemic complications, with high-fat diets (HFDs) playing a key role by disrupting intestinal balance and promoting oxidative stress. This study investigates Protaetia brevitarsis-derived protein hydrolysate (PBPH), an insect-derived bioactive peptide extract, as a potential intervention to counteract HFD-induced metabolic disturbances. Female ICR mice were divided into three groups: control diet, HFD, and HFD + PBPH, with PBPH (obtained by alcalase hydrolysis and ultrafiltration) administered daily for eight weeks. Researchers assessed adipokine levels, inflammatory cytokines, antioxidant enzymes, and apoptotic markers using qPCR, ELISA, histology, and immunohistochemistry. PBPH supplementation significantly improved metabolic parameters by lowering leptin, adipsin, resistin, IL-1β, TNF-α, and IL-6, while restoring antioxidant balance and reducing pro-apoptotic signals. Histological analyses confirmed preserved intestinal tissue and reduced inflammation. Overall, this study highlights PBPH's promising therapeutic role in addressing obesity-related metabolic dysfunctions through its multifaceted effects on inflammation, oxidative stress, and apoptosis. It underscores the potential of insect-derived peptides as sustainable, innovative dietary interventions for improving metabolic health.

Indexed as

Adipose TissueAnti-Inflammatory AgentsAntioxidantsObesityProtein HydrolysatesRhodophytaAnimalsCytokinesDiet, High-FatFemaleInflammationMiceMice, Inbred ICRMice, ObeseOxidative StressAnti-Inflammatory AgentsAntioxidantsCytokinesProtein Hydrolysatesanti-inflammatoryanti-oxidativehigh fat dietobesityprotaetia brevitarsis-derived protein hydrolysate

Identifiers

PMID41226391
PMCPMC12607786

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.