ArticleProceedings of the National Academy of Sciences of the United States of America2025
SERK5 genetically compensates for the loss of BAK1 and BKK1 in regulating elf18-triggered immune signaling in
Article in Proceedings of the National Academy of Sciences of the United States of America, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Authors and funding
7 authors.
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Abstract
SOMATIC EMBRYOGENESIS RECEPTOR-LIKE KINASEs (SERKs), particularly BRASSINOSTEROID INSENSITIVE1-ASSOCIATED KINASE 1 (BAK1) (SERK3) and BKK1 (SERK4), function as pleiotropic coreceptors to transduce extracellular developmental and immune signals in
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