ArticleInflammopharmacology2025
Myricetin alleviates indomethacin-induced gastric ulcers in rats via antioxidant, anti-inflammatory and antiapoptotic mechanisms.
Article in Inflammopharmacology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
3 citing papers in PubMed.
- The synergistic protective effects of bioactive catechins in longjing tea: alleviate indomethacin-induced gastric toxicity through modulation of inflammatory and apoptotic pathways.Frontiers in pharmacology · 2026Article
- Diabetic foot osteomyelitis: pathological microenvironment and matching tailored treatment strategies.Frontiers in cell and developmental biology · 2026Review
- Nrf2/Keap1 Signaling Axis in the Brain: Master Regulator of Oxidative Stress in Neurodegenerative and Psychiatric Disorders.Molecular neurobiology · 2025Review
Corrections and comments
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Authors and funding
6 authors.
Funding
Abstract
Gastric ulcers are among the most widespread gastrointestinal diseases, often caused by NSAIDs. Natural compounds with less toxicity and more safety are being explored to minimize their impact. The main goal is to examine the gastroprotective effects of Myricetin in indomethacin-induced gastric ulcers in rats, and the mechanism involved. The rats were allocated into control, negative, standard, Myricetin 10 mg/kg, and Myricetin 20 mg/kg groups. The effect of Myricetin on indomethacin-induced ulcers was examined for 14 days. The Ulcer index, gastric acidity, and PGE
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Registered trials
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