ArticleProceedings of the National Academy of Sciences of the United States of America2025
Exploring evolutionary trajectories of drug resistance.
Article in Proceedings of the National Academy of Sciences of the United States of America, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
1 citing paper in PubMed.
- Generative Artificial Intelligence-Empowered Virtual Evolution of Enzyme with the VERnet Model.ACS catalysis · 2026Article
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3 authors.
Funding
Abstract
Drug resistance poses a major global health challenge, necessitating the development of effective therapeutic strategies. The main challenge is to predict drug-resistant mutations and design drugs that retain efficacy against such evolving targets. Our previous effort in computing the vitality value has provided a framework in assessing drug resistance. While promising, the approach lacked accuracy due to insufficient information about mutation tendencies and protein stability. In this study, we used the Stanford University HIV Drug Resistance Database and observed that drug resistance, usually quantified as [Formula: see text], exhibits a positive correlation with the Maximum Entropy energy, [Formula: see text]. However, both drug resistance and vitality are also correlated with the number of mutations, indicating that the virus cannot easily gain resistance through specific mutational pathways and must sacrifice stability and function to escape inhibition. To overcome this number dependence, we looked for a system with less extensive mutagenesis and chose HCV protease. In this case, resistance substitutions cluster at low [Formula: see text] values, reflecting a limited mutational space. This restricted landscape enables [Formula: see text] to predict evolutionary pathways of resistance and to guide the identification of robust therapeutic candidates.
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Registered trials
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