Evidence map›Paper›PMID 41169388›Full record

ArticleFrontiers in immunology2025

Luise Ehlers, Anika Kasprick, Ottavia Agrifoglio, Natalie Gross, Nancy Ernst, Alanis Barbosa Gulde, Colin Osterloh, Noa Linn Brauckmann, Nicole Y Fischer, Wendy Bergmann-Ewert and 3 more

Abstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Frontiers in immunology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

13 authors.

Luise EhlersDepartment of Internal Medicine, Division of Gastroenterology, Hepatology and Nutritional Medicine, Rostock University Medical Center, Rostock, Germany.
Anika KasprickLübeck Institute of Experimental Dermatology and Center for Research on Inflammation of the Skin, University of Lübeck, Lübeck, Germany.
Ottavia AgrifoglioDepartment of Internal Medicine, Division of Gastroenterology, Hepatology and Nutritional Medicine, Rostock University Medical Center, Rostock, Germany.
Natalie GrossLübeck Institute of Experimental Dermatology and Center for Research on Inflammation of the Skin, University of Lübeck, Lübeck, Germany.
Nancy ErnstLübeck Institute of Experimental Dermatology and Center for Research on Inflammation of the Skin, University of Lübeck, Lübeck, Germany.
Alanis Barbosa GuldeLübeck Institute of Experimental Dermatology and Center for Research on Inflammation of the Skin, University of Lübeck, Lübeck, Germany.
Colin OsterlohLübeck Institute of Experimental Dermatology and Center for Research on Inflammation of the Skin, University of Lübeck, Lübeck, Germany.
Noa Linn BrauckmannLübeck Institute of Experimental Dermatology and Center for Research on Inflammation of the Skin, University of Lübeck, Lübeck, Germany.
Nicole Y FischerDepartment of Internal Medicine, Division of Gastroenterology, Hepatology and Nutritional Medicine, Rostock University Medical Center, Rostock, Germany.
Wendy Bergmann-EwertCore Facility for Cell Sorting and Cell Analysis, Rostock University Medical Center, Rostock, Germany.
Ralf J LudwigLübeck Institute of Experimental Dermatology and Center for Research on Inflammation of the Skin, University of Lübeck, Lübeck, Germany.
Katja BieberLübeck Institute of Experimental Dermatology and Center for Research on Inflammation of the Skin, University of Lübeck, Lübeck, Germany.
Robert JasterDepartment of Internal Medicine, Division of Gastroenterology, Hepatology and Nutritional Medicine, Rostock University Medical Center, Rostock, Germany.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Background: Autoimmune pancreatitis (AIP) is a multifactorial disease caused by both genetic and environmental factors. Previous studies have implicated Bach2, a key regulator of adaptive immunity, in the pathogenesis of this disease. However, direct experimental evidence is lacking. Here, we used C57BL/6N mice with a targeted deletion of Methods: In this multicenter preclinical study, Results: Consistent with the results of previous studies, Conclusion:

Indexed as

Autoimmune PancreatitisBasic-Leucine Zipper Transcription FactorsDiet, High-FatFatty LiverAnimalsDisease Models, AnimalMaleMiceMice, Inbred C57BLMice, KnockoutPoly I-CBach2 protein, mouseBasic-Leucine Zipper Transcription FactorsPoly I-Cautoimmune pancreatitisBach2fatty liver diseasehigh-fat dietmouse model

Identifiers

PMID41169388
PMCPMC12568605

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.